• The addition of epinephrine to local anesthetic solutions produces vasoconstriction.

    局麻药溶液加入肾上腺素,使血管收缩

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  • Both acute or chronic hypoxia can cause vasoconstriction and vasoconstriction increase.

    急性慢性低氧都能使肺血管收缩,血流阻力增大

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  • ET might be the important factor to cause vasoconstriction, resulting in the portent symptoms.

    ET可能导致血管收缩引起先兆症状重要因子

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  • Results The intrarenal vasoconstriction in different stages of cirrhosis was significantly difference.

    结果不同阶段肝硬化患者肾内血管收缩程度明显的差异。

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  • The contraction calf muscle, urges the vein vasoconstriction, the acceleration vein blood backflow heart.

    收缩小腿肌肉促使静脉血管收缩加速静脉血液回流心脏

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  • AIM: to investigate the effects of platelet-derived growth factor BB (PDGF-BB) on vasoconstriction of the vascular vessels.

    目的观察血小板衍生生长因子-BB (PDGF - BB)大鼠主动脉血管收缩作用

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  • Somatostatin and analogues such as octreotide and vapreotide also cause splanchnic vasoconstriction at pharmacological doses.

    生长抑素及其类似物奥曲普肽等药理剂量下可以引起内脏血管收缩

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  • Furthermore, ECs obtained from these vascular malformations can be used as an experimental model to study SAH-induced vasoconstriction.

    此外这些来源于畸形血管内皮细胞作为研究蛛网膜下腔出血引发血管收缩实验模型

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  • However, in hypoxic pulmonary hypertension rats, 5 - HT - induced vasoconstriction is mediated by both the 5 - HT1Band 5 - HT2A receptor.

    但是慢性低氧肺动脉高压大鼠5-HT诱导血管收缩反应通过5 -HT _ (2 A)和5 - HT _ (1 B)受体介导的。

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  • Endothelin is one of the most potent vasoconstriction peptides, and it plays important physiological role by interacting with its receptors.

    内皮目前已知缩血管活性最强的生物活性之一,通过与内皮素受体结合发挥广泛的生物学活性。

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  • Hypoxia produced a sustained increase in pulmonary arterial pressure (Ppa), the site of vasoconstriction was proximal to the lung capillaries.

    缺氧时肺动脉显著升高可能毛细血管前肌动脉收缩的结果。

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  • Pulmonary vascular smooth muscle cell is an important executor of vasoconstriction and a key contributor of pulmonary vascular structure remodeling.

    血管平滑肌细胞肺血管收缩反应主要执行者,也是肺血管结构重建的重要参与者

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  • Objective To study the effects of different concentration of desflurane on hypoxic pulmonary vasoconstriction (HPV) during one-lung ventilation (OLV).

    目的观测地醚麻醉深度单侧肺通气(OLV)时低血管收缩(HPV)的影响

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  • Conclusion Reduced vasodilator reserve or inappropriate vasoconstriction of the coronary microvessels are implicated as causes of microvascular angina.

    结论微血管心绞痛由于小冠状动脉扩张贮备降低异常收缩导致心肌缺血。

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  • Abnormal vasoconstriction of digital arteries and cutaneous arterioles due to a local defect in normal vascular responses is thought to underlie the disorder.

    本病基础是正常血管反应局部缺陷而导致的指(趾)动脉皮肤动脉异常血管收缩。

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  • Nicotine causes vasoconstriction, a narrowing of the blood vessels that can limit oxygen-rich blood flow to the tiny vessels in the face or other parts of the body.

    尼古丁会导致血管收缩,血管收缩将限制富氧血液流向面部身体其他部位细微血管

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  • The inhibition of prostaglandin production with Ibuprofen strengthened hypoxic pulmonary vasoconstriction thus improved pulmonary gas exchanges in acid lung injury.

    布洛芬抑制前列腺生成,并通过增强缺氧性血管收缩反应而改善了损伤时的气体交换

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  • When acute anoxia, the skin, the abdominal cavity internal organs nerve is excited, shrinks the blood vessel function to get the advantage, therefore vasoconstriction;

    急性缺氧时皮肤腹腔内脏交感神经兴奋血管作用占优势血管收缩

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  • You can tell when stress hormones are acting since one also develops cold hands and cold feet during the day from the vasoconstriction of the blood vessels to the hands and feet.

    应激激素发挥作用能够感觉到因为会引发血管收缩以至于白天会有手脚冰凉的现象。

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  • METHODS Isometric vasoconstriction of the rabbit saphenous arterial rings was recorded, and the sympathetic nerves of the arterial rings were activated with electrical stimulation.

    方法动脉血管张力记录法电场刺激诱发血管收缩法。

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  • The drugs' mechanism of action remains uncertain, but plausible theories include vasoconstriction, modulation of pro-survival signal transduction pathways, and endothelial cell apoptosis.

    药物作用机制不清楚,可能的作用包括血管收缩调节信号传导通路促进内皮细胞凋亡

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  • The basic pathological alteration of pulmonary hypertension involves three dependent elements:pulmonary vasoconstriction, pulmonary vascular remodeling and pulmonary microvascular thrombogenesis.

    前言:肺动脉高压基本病理改变方面血管收缩、肺血管重构肺小血管内微血栓形成。

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  • The basic pathological alteration of pulmonary hypertension involves three dependent elements:pulmonary vasoconstriction, pulmonary vascular remodeling and pulmonary microvascular thrombogenesis.

    前言:肺动脉高压基本病理改变方面血管收缩、肺血管重构肺小血管内微血栓形成。

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