这类代谢药物主要通过诱导游离脂肪酸代谢向葡萄糖代谢的转换，从而增加单位氧耗生成三磷酸腺苷供能物质的量而发挥抗心绞痛作用。
These metabolic drugs induce a shift from free fatty acid towards predominantly glucose utilisation by the myocardium to increase ATP generation per unit of oxygen consumption.
这类代谢药物主要通过诱导游离脂肪酸代谢向葡萄糖代谢的转换，从而增加单位氧耗生成三磷酸腺苷供能物质的量而发挥抗心绞痛作用。
These metabolic drugs induce a shift from free fatty acid towards predominantly glucose utilisation by the myocardium to increase ATP generation per unit of oxygen consumption.
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