The active ingredients can target at many aspects of brain injury after hypoxia-ischemia.
中药的活性成分能多靶点地作用于脑缺血缺氧后的损伤环节。
RESULTS: All the 148 cases of hypoxia-ischemia brain injury were involved in the result analysis.
结果纳入缺氧缺血性脑损伤患儿148例，全部进入结果分析，无脱落。
Effects of Pretreatment with HBO on Brain SOD Activity and MDA Content following Rats Hypoxia-Ischemia in Uterus.
高压氧预处理对宫内缺氧缺血大鼠脑组织SOD活性、MDA含量的影响。
The animal models of cerebral hypoxia-ischemia were prepared in 7 days old rats by systemic hypoxia for 2h and right common carotid artery ligation.
结扎并低氧处理7日龄大鼠右侧颈总动脉制备半球性脑缺血损伤模型。
Serum samples taken from 22 patients with hypoxia-ischemia encephalopathy (HIE were determined with enzyme immunoassay using a kit for the measurment of NSE.
本文采用酶免疫分析法测22例缺氧缺血性脑病（HIE患儿血清中神经元特异性烯醇化酶（NSE的变化。
Ischemia and hypoxia induce the death of nerve cells and discontinue of the nerve fiber.
缺血、缺氧导致神经细胞死亡和神经纤维中断。
Some local factors to destroy intestine crest cells such as ischemia, hypoxia, immune factors, Epstein-Barr virus and parasite infection.
某些局部因素。如缺血、缺氧、免疫因素、巨细胞病毒和寄生虫感染等毁坏了肠壁内神经嵴细胞。
The research of the mechanism involves in biochemistry, molecular biology and blood dyscrasia, but it's center still is high blood sugar which causes a series changes of ischemia and hypoxia.
发病机制的研究涉及生物化学、分子生物学、血液异常的等方面，但其中心环节是高血糖和其引起组织缺血缺氧发生的一系列改变。
Conclusion: the changes of N1P1 in acute attack period are related with the neuron toxic action of excitatory amino acids and lesions made by secondary ischemia and hypoxia.
结论：癫痫急性发作期n 1、P 1波的改变与兴奋性氨基酸的神经元毒性作用及继发性缺血、缺氧致神经元损伤有关。
BACKGROUND: brain injury often causes secondary cerebral ischemia and hypoxia, which aggravate the brain damages.
背景：脑损伤后常引起继发性脑缺血、缺氧，使脑损害加重。
Renal injury after ischemia appears to be a consequence of tissue hypoxia not only from interrupted blood supply but also from the process of reperfusion which leads to an active inflammatory process.
肾缺血后损伤显示，损伤不仅是血供中断后组织缺氧的结果，更有再灌注进程中引起炎症反应的激活过程。
The indices that reflecting acute hypoxia were ranked as follows: pulse rate, myocardial oxygen consumption, coronary ischemia threshold, cardiac output and car-diac index.
研究还发现，反映急性缺氧变化敏感的指标依次是脉率、心肌耗氧量与冠脉缺血阈值，以及心输出量与心脏指数。
ConclusionThe important pathological changes in the placenta of PIH are placental ischemia and hypoxia, leading to proliferation of trophoblastic cells and damage of vascular endothelial cells.
结论妊高征重要的病理学改变为胎盘缺血、缺氧致滋养层细胞增生及血管内皮细胞损伤。
METHOD to determine the effect of the preparations on induced myocardial ischemia, myocardial ischemia due to ligation of the main coronary artery, ischemic shock and hypoxia.
方法测定对诱发性心肌缺血、失血性冠状动脉主干结扎性心肌缺血、失血性休克、耐缺氧的影响。
Glucose transport albumen (GLUT-1) is a kind of carrier controlling glucose transport existed widely in human organism, which can have increased expression under the condition of hypoxia and ischemia.
葡萄糖转运蛋白1是广泛存在于人体各组织的细胞控制葡萄糖转运的载体，在缺氧及缺血状态下可以出现表达增高。
Conclusion Propofol may inhibit hypoxia in the brain and the apoptosis of nerve cells in result of protecting the cerebral ischemia and reperfusion damage in rats.
结论异丙酚可能通过抑制大脑缺氧，抑制神经元的凋亡，从而对大鼠局灶性脑缺血-再灌注损伤发挥保护作用。
Objective: To establish a novel hypoxia ischemia model of animal.
目的：建立可靠的缺氧缺血动物模型。
High sustained + Gz acceleration produced by modern high performance aircrafts can induce brain damage because of ischemia and hypoxia.
现代高性能战斗机产生的高g值持续性正加速度可导致脑缺血、缺氧，继而引起脑功能障碍。
Conclusion Mailiping has protective effect on myocardial ischemia and hypoxia.
结论麦利平对心肌缺血缺氧具有保护作用。
Objective: Explore the effects of YTG on red cells deformation ability of rat with cerebral ischemia and mouse's decompression hypoxia ability.
目的：探讨养阴通脑颗粒对脑缺血大鼠红细胞变形能力和小鼠减压缺氧耐力影响。
AIM To study the effect of CGRP on L-type calcium channel current in guinea pig ventricular myocytes under normal and imitating ischemia and hypoxia conditions.
目的探讨降钙素基因相关肽(CGRP)对正常及模拟缺血缺氧状态下豚鼠心肌细胞钙通道电流的作用。
Conclusion Acute low temperature and hypoxia may make damages of myocardial ischemia rabbits much severe.
结论急性低温复合低氧对心肌缺血的家兔有加重心肌缺血作用。
Objective To study the effects of acute low temperature and hypoxia on serum SOD and MDA of myocardial ischemia rabbit.
目的探讨心肌缺血的家兔在急性低温低氧复合环境下血清超氧化物歧化酶（SOD）活力和丙二醛（MDA）含量的变化。
Conclusions Opening ETS can cause cerebral ischemia and hypoxia during intracranial hypertension.
结论颅内高压状态下进行开放式ETS可引起脑缺血、缺氧反应。
"Xinjining Capsule" could reverse the changes of myocardial electrophysiology resulted from ischemia-hypoxia, but Amiodarone had no such effect.
心疾宁能逆转缺血低氧引起的心肌细胞电生理改变，而胺碘酮无此作用。
Results Hemorrhagic shock, systemic changes including ischemia and hypoxia, energy shortage may lead to secondary diabetes insipidus.
结果各种原因所致失血性休克、全身性缺血缺氧改变、能量供给障碍等均可能导致继发性尿崩症。
Autophagy plays an important role in a variety of life activity. For example, it can speed up cell metabolism or have the cells obtained energy from the decomposition products under ischemia-hypoxia.
自噬在各种生命活动中发挥着重要作用，例如它可以加速细胞内的新陈代谢，或者在细胞处于缺血缺氧等饥饿状态时从分解产物中获得能量。
Preterm newborn child suffering from epilepsy in a higher proportion of perinatal asphyxia, chronic functional brain hypoxia, congenital dysplasia, and so is the cause of cerebral ischemia.
新生儿中早产儿患上癫痫病的比例较高，围产期窒息、慢性功能性脑缺氧、先天性脑发育不良、脑缺血等是致病原因。
The model of mice with acute myocardial ischemia was made by subcutaneous injection of Iso in order to observe the survival time in normobaric hypoxia of mice in each group.
②对异丙肾上腺素所致急性心肌缺血小鼠缺氧存活时间的影响：采用异丙肾上腺素皮下注射制备小鼠急性心肌缺血模型，观察各组小鼠常压耐缺氧存活时间。
Conclusions When the brain is severely injured, there is cerebral hypoxia or ischemia during the first 24 hours, and cerebral hyperemia in the following second-fourth days.
结论重型脑损伤后24小时内存在脑缺氧、缺血，伤后第2 - 4天为脑氧合过度、脑充血。
应用推荐