Intestinal bacterial translocation is the prime source of pancreas infection.
胰腺感染细菌的来源主要是肠道细菌的移位。
Simultaneous increase in intestinal permeability, intestinal flora imbalance, intestinal bacterial translocation.
同时出现肠道通透性增加，肠道菌群失调，肠道细菌易位。
Thus, it is considered that extrahepatic biliary obstruction may induce intestinal bacterial translocation in man.
提示肝外胆道梗阻可导致人体肠道细菌易位。
Objective:To observe intestinal bacterial translocation in acute necrotizing pancreatitis (ANP), and to elucidate whether the gut would be the source of bacteria in pancreatic infection.
目的：研究急性坏死性胰腺炎（ANP）时肠道细菌易位情况，探讨肠道是否为继发性胰腺感染的细菌来源。
Histamine; Aged rats; Intestinal mucosa barrier; Bacterial translocation.
组胺；老龄大鼠；肠黏膜屏障；肠道细菌易位。
The indexes observed were intestinal transmit index, bacterial translocation rate, serum amylase, histological score of the pancreas and the level of D lactate.
检测指标包括：肠道转运系数、血清淀粉酶、脏器细菌移位率、胰腺病理评分、血浆D 乳酸等。
The key to prevent dysbacteriosis lies in rational use of antibiotics, ensuring the integrity of intestinal mucosal barrier so as to avoid bacterial translocation.
预防菌群失调症的关键在于合理使用抗生素，保证肠道黏膜屏障的完整性，预防肠道细菌移位。
The intestinal morphology, permeability of intestinal mucosa, bacterial translocation and gut immune barrier function were compared.
观察肠道形态学、肠道黏膜通透性、肠道细菌易位情况和血浆内毒素水平及肠道免疫功能检测。
Intestinal flora including Bifidobacteria, Lactobacilli, Enterobacteriaceae and Clostridium perfringens in the feces and bacterial translocation in mesenteric lymph nodes and spleens were detected.
以小鼠粪便中的双歧杆菌、乳酸杆菌、肠杆菌和产气荚膜梭菌数量及肠系膜淋巴结、脾脏细菌移位为检测指标。
Purpose To evaluate the effect of cisapride on the intestinal bacterial flora and the incidence of bacterial translocation in cirrhotic rats.
目的观察肝硬化大鼠肠道细菌过度生长和肠道细菌转位情况及西沙必利对其影响。
Conclusion LP administration can modulate the imbalance of intestinal flora and decrease the bacterial translocation, thus enhance intestinal barrier function in mice with IBD.
结论LP能纠正炎症性肠病小鼠肠道菌群紊乱，减少细菌移位，从而增强了肠道屏障功能。
Conclusion LP administration can modulate the imbalance of intestinal flora and decrease the bacterial translocation, thus enhance intestinal barrier function in mice with IBD.
结论LP能纠正炎症性肠病小鼠肠道菌群紊乱，减少细菌移位，从而增强了肠道屏障功能。
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