Methods: keloid fibroblasts were isolated from fresh keloid tissue and cultured.
方法取新鲜瘢痕疙瘩组织，体外培养瘢痕成纤维细胞。
Conclusion P53 can obviously inhibit MMP1 and MMP3 expression in human keloid fibroblasts.
结论P 53蛋白能够抑制人瘢痕疙瘩成纤维细胞MMP1、MMP3蛋白的表达。
Conclusion: P53 protein significantly inhibits telomerase activity in human keloid fibroblasts.
结论：P 53蛋白能够抑制人瘢痕疙瘩成纤维细胞端粒酶活性。
Conclusion Chitosan can inhibit the growth, proliferation of fibroblasts derived from keloid, hypertrophic scars and normal skin, and may play important role in the prevention and treatment of scars.
结论：几丁糖对正常皮肤及增生性瘢痕和瘢痕疙瘩来源的成纤维细胞的生长增殖均有抑制作用，有望在瘢痕的防治中发挥重要作用。
METHODS: Keloid and norm al skin tissue were collected for the fibroblasts in vitro using tissue-block cultivation.
方法：取瘢痕疙瘩和正常皮肤，采用组织块培养法进行成纤维细胞体外培养。
Conclusion the recombinant adenovirus vector carrying human TRADD gene was successfully constructed. It can be used for detecting the effects of TRADD on fibroblasts of hypertrophic scar and keloid.
结论构建成功的含有TRADD基因片段的重组腺病毒，可用于转染人成纤维细胞，用于检测TRADD对病理性瘢痕成纤维细胞的影响。
Conclusion the recombinant adenovirus vector carrying human TRADD gene was successfully constructed. It can be used for detecting the effects of TRADD on fibroblasts of hypertrophic scar and keloid.
结论构建成功的含有TRADD基因片段的重组腺病毒，可用于转染人成纤维细胞，用于检测TRADD对病理性瘢痕成纤维细胞的影响。
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