Aim Establish a novel mouse model of airway goblet cell hyperplasia and mucus hypersecretion.
目的建立一种新的小鼠气道上皮杯状细胞增生和粘液高分泌模型。
Lung tissues were examined for cell infiltration and mucus hypersecretion, and the expression of inflammatory biomarkers.
取肺组织检测细胞浸润、粘液的过度分泌以及炎症性生物标记物的表达。
Mucus hypersecretion in airway is the common pathophyisolo gi cal characteristic of many chronic airway inflammation diseases.
目的：介绍慢性炎症气道粘液高分泌的治疗进展。
Objective To investigate the roles of theaflavins (TFs) in airway mucus hypersecretion induced by human neutrophil elastase (HNE).
目的观察茶黄素(TF)各单体在人中性粒细胞弹性蛋白酶(HNE)诱导气道黏液高分泌中的作用。
Conclusion This model helps to screen the new drugs for mucus hypersecretion and research on the pathological mechanism of airway mucus hypersecretion.
结论这一模型有助于筛选抗黏膜高分泌的新药和研究气道黏膜高分泌的病理机制。
Background and Objective Asthma is chronic airway immune inflammation characterized by airway lymphocyte infiltration and eosinophilia, airway hyperresponsiveness (AHR) and mucus hypersecretion.
背景与目的哮喘是慢性气道免疫性炎症，以气道淋巴细胞、嗜酸性粒细胞浸润、气道高反应性、粘液产生增加为特征。
Background and Objective Asthma is chronic airway immune inflammation characterized by airway lymphocyte infiltration and eosinophilia, airway hyperresponsiveness (AHR) and mucus hypersecretion.
背景与目的哮喘是慢性气道免疫性炎症，以气道淋巴细胞、嗜酸性粒细胞浸润、气道高反应性、粘液产生增加为特征。
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