ACEI has a regulating role for cytokine levels and myocardial fibrosis in patients with CHF.
ACEI对CHF患者促炎性细胞因子和心肌纤维化水平具有调控作用。
The myocardial fibrosis was characterized by cardiac collagen morphological quantitive analyses.
心肌纤维化的改变采用心肌胶原形态定量分析。
ObjectiveTo observe the effects of resveratrol on myocardial fibrosis in atherosclerotic rabbits.
目的观察白藜芦醇对动脉粥样硬化兔心肌纤维化的影响。
This paper reviews the study about myocardial fibrosis in its pathogenesis, prevention and treatment.
本文就心肌纤维化的发病机制及防治的研究进展简要综述。
Objective To investigate the expression of cycooxygenase 2(COX2) in myocardial fibrosis and significant.
目的：研究环氧合酶2（COX2）在心肌纤维化中的表达及意义。
Collagen of the heart was stained by Sirius red to observe the effects of the drugs on myocardial fibrosis.
天狼星红胶原染色大鼠的心脏切片，观察药物干预对心肌纤维化的影响。
Studies manifested that CTGF was associated with myocardial fibrosis in the rat model of myocardial infarction.
多个研究发现，在鼠的心肌梗死模型CTGF与心肌梗死后心肌纤维化密切相关。
This article narrates characteristic, mechanism, serolog and drug inversion of myocardial fibrosis in hypertension…
现就高血压心肌纤维化的特点、机制、检测指标以及药物逆转效应作一综述。
OBJECTIVE To observe the effect of Taohe Chengqi Decoction and different extractions on myocardial fibrosis of rats with diabetes.
目的观察加味桃核承气汤及不同提取物对糖尿病大鼠心肌纤维化的影响。
The effect of regressing myocardial fibrosis in the rat with CHF by captopril may be related to the decrease in plasma aldosterone level.
卡托普利逆转压力负荷增加大鼠心肌纤维化的作用可能与降低血浆醛固酮水平有关。
The abnormal accumulation of interstitial collagen and myocardial fibrosis could destroy the network structure of myocardial interstitium.
并能使心肌组织电的各向异性增加，容易引起心律失常，甚至猝死。
Objective: To observe the protective effects of puerarin on cardiac muscle in diabetic rats and explore the mechanism of myocardial fibrosis.
目的：观察葛根素对糖尿病大鼠心肌损伤的保护作用，探讨心肌纤维化可能的发病机制。
Stained the collagen in the slice of the heart by Sirius red to observe the effects of the drugs on vessel remodeling and myocardial fibrosis.
天狼星红胶原染色大鼠的心脏切片，观察药物干预对血管重塑和心肌纤维化的影响。
This paper summarizes the detection of myocardial fibrosis in laboratory, and the progress and mechanisms of medication in myocardial fibrosis.
本文综述了心肌纤维化的实验室检测以及多种药物治疗心肌纤维化的进展及其作用机制。
The increase of circulating catecholamines exerts direct toxic effect on cardiac myocytes, increases membrane permeability and myocardial fibrosis;
循环儿茶酚胺升高对心肌细胞产生直接的毒性作 用、增加膜通透性和心肌纤维化，这些作用可引起细胞死亡。
Myocardial fibrosis in both diabetic and non-diabetic rats subjected to AMI was similar in the early phase, while it was quite different after 1 month.
在早期阶段，糖尿病与非糖尿病大鼠心肌纤维化相似，而1月后却出现显著差别。
Objective: Myocardial fibrosis is one of the important cause of some end-stage heart disease such as chronic heart failure, arrhythmia and even sudden cardiac death.
目的：心肌纤维化是慢性心力衰竭、心律失常甚至心源性猝死等心脏疾病的重要原因。
Myocardial fibrosis is caused by overdeposition of myocardial interstitial collagen and proportion disorder of all kinds of collagen, and it occurs in many heart diseases.
心肌纤维化是由于心肌间质胶原沉积增多，各型胶原比例失调所导致，可发生于多种心血管疾病。
Objective To explore whether the Astragalus injection (AI) has effect for reversing left ventricular hypertrophy and myocardial fibrosis induced by renal vascular hypertension in rats.
目的探讨黄芪注射液对肾血管性高血压大鼠左心室肥厚及心肌纤维化有无逆转作用。
Purpose To study the effects of myocardial fibroblasts on fibrosis in diabetic cardiomyopathy.
目的研究成纤维细胞在糖尿病心肌纤维化病变中的作用。
Myocardial oxidative stress and thiol-sensitive signaling molecules are implicated in pathogenesis of hypertrophy and fibrosis.
心肌氧化应激和硫醇敏感的信号转导分子参与心肌肥厚和纤维化的发病过程。
CONCLUSION: Myocardial interstitial fibrosis and the reduction of myofibril elements are the major pathological basis for the impairment of cardiac function in patients with DCM.
结论：DCM患者心肌间质纤维化，肌原纤维减少是扩张型心肌病心功能损害的主要病理基础。
Electromicroscopy pointed to myocardial interstitial fibrosis and MC infiltration, whose presence in cirrhotic myocardium may have played a role in the pathogenesis of cirrhotic cardiomyopathy.
结果表明肝硬化存在心肌间质纤维化和肥大细胞浸润，可能参与肝硬化心肌病的发生。
ACEI reduces the levels of ACEmRNA directly and significantly inhibits the progression of myocardial interstitial fibrosis and cardiac remodeling.
臼acei可直接下调ace测a表达，有效地抑制了心肌间质纤维化，阻止心肌重塑的发生。
Objective To study the changes of tissue fibrosis indexes in serum and to evaluate their clinical value in reflecting the process of fibrosis in aged patients with previous myocardial infarction (MI).
目的通过对老年陈旧性心肌梗死（MI）患者组织纤维化血清指标变化的观察，探讨将其作为反映MI患者心肌纤维化过程指标的临床应用价值。
Objective To investigate the mechanism of reversion of myocardial interstitial fibrosis in diabetic cardiomyopathy (DCM) by valsartan.
目的探讨缬沙坦逆转糖尿病心肌病(DCM)心肌间质纤维化的作用机制。
CONCLUSION Fluvastatin could inhibit myocardial interstitial fibrosis and improve the cardiac function of diabetic rats, which might be related to the down-regulating of the CTGF expression.
结论氟伐他汀可逆转糖尿病大鼠心肌间质纤维化并改善心功能，这种作用可能与其抑制CTGF的表达有关。
The myocardium beneath the endocardial surface at the top demonstrates pale fibrosis with collagenization following healing of a subendocardial myocardial infarction.
图上方心内膜下的心肌可见苍白色纤维化，是心内膜下心肌梗死治疗后形成的。
The myocardium beneath the endocardial surface at the top demonstrates pale fibrosis with collagenization following healing of a subendocardial myocardial infarction.
图上方心内膜下的心肌可见苍白色纤维化，是心内膜下心肌梗死治疗后形成的。
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