The causes of degeneration and death of the SGN in acoustic trauma are as yet not completely known.
噪声通过机械、代谢和血运等途径损伤毛细胞的机制已基本明确,但SGN的变性死亡的原因还不清楚。
There was a possibility of acoustic trauma in the artillerymen with late—onset tinnitus or deafness.
晚期出现耳聋、耳鸣症状的炮兵,有听力损伤的潜在可能。
Conclusion: Prophylactic inhalation of negative air ions can increase the cochlear blood flow and promote microcirculation thus protecting the cochlea from acoustic trauma.
结论:预防性吸入空气负离子可增加耳蜗血流量,改善微循环,因而对保护噪声性耳蜗损伤是有益的。
Objective: to observe the protective effect of sound preconditioning on hearing in acoustic trauma and explore the relevant mechanism with regard to free oxygen radicals in the cochlea.
目的:观察声预处理对噪声性听力损失的保护作用,并从氧自由基途径探讨其机制。
Objective: to observe the protective effect of sound preconditioning on hearing in acoustic trauma and explore the relevant mechanism with regard to free oxygen radicals in the cochlea.
目的:观察声预处理对噪声性听力损失的保护作用,并从氧自由基途径探讨其机制。
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