CONCLUSION: EAAC1 antisense oligonucleotide has the neuroprotective effect against acute cerebral ischemia injury.
结论：EAAC 1反义寡核苷酸对急性脑缺血损伤有神经保护作用。
Objective To evaluate the protective effect of scopolamine against acute cerebral ischemia reperfusion injury.
目的探讨东莨菪碱对急性脑缺血再灌注损伤的保护作用。
BACKGROUND: Cell apoptosis and expression of related apoptotic gene are found in rats with acute cerebral ischemia-reperfusion injury.
背景：大鼠急性脑缺血再灌注损伤后有细胞凋亡及凋亡相关基因的表达。
Results:No perioperative death, no spinal cord injury, limb ischemia, stent migration, serious complications such as acute cerebral ischemia.
结果：全组无围术期死亡，无脊髓损伤、肢体缺血、支架移位及急性脑缺血等严重并发症。
METHODS: Pulsinelli four vessel occlusion method was used to make acute cerebral ischemia reperfusion injury in Wistar rats.
方法：采用四动脉结扎法，制作大鼠全脑缺血再灌注模型。
Objective To study the protective effect of sibelium to acute cerebral ischemia reperfusion injury.
目的进一步探讨西比灵对脑缺血再灌注损伤保护作用的机制。
METHOD The method of ligating both common carotids and vagus was used to make acute cerebral ischemia reperfusion injury in mice.
方法采用小鼠双侧颈总动脉和迷走神经结扎法制作脑缺血模型并观察脑舒宁对其的影响。
METHOD The method of ligating both common carotids and vagus was used to make acute cerebral ischemia reperfusion injury in mice.
方法采用小鼠双侧颈总动脉和迷走神经结扎法制作脑缺血模型并观察脑舒宁对其的影响。
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