Objective Massive necrosis and apoptosis of liver cells is the common feature of different kinds of liver diseases, such as viral hepatitis, alcoholic hepatitis, autoimmune hepatitis, etc.
目的：在自身免疫性肝炎和病毒性肝炎引起的肝损伤发病过程中，肝细胞的坏死和凋亡是共同特点。
SA induced apoptosis in human liver cancer cells, which may be one of the mechanisms of its antitumor effects.
诱导人肝癌细胞凋亡，可能是其抗肿瘤作用机制之一；
Conclusion: Ammonia may induce the histopathological changes of the brain in rats with liver cirrhosis and the apoptosis of brain cells may be the foundation of these changes.
结论：氨可能诱发肝硬化大鼠脑发生形态变化，脑神经细胞凋亡可能是这种变化的基础。
BACKGROUND&AIM: To study the effect of holmium ions on DNA breakage of mice liver cells and the effect of holmium element on apoptosis of zoic cells in vivo.
背景与目的： 通过研究钬离子溶液对小鼠肝脏细胞DNA的损伤，探讨钬元素对诱导动物细胞凋亡的影响。
These results indicated that both the infected and uninfected liver cells could occur apoptosis in patients with viral hepatitis. Further study on the mechanism of liver cell apoptosis is needed.
提示病毒性肝炎患者感染和未感染病毒的肝细胞均可发生凋亡，其机制有待进一步研究。
PEG10 gene accelerated the growth of L02 cells and inhibited their apoptosis but it had no conspicuous effect on the non-liver derived cells.
该质粒经过稳定筛选后稳定表达于细胞内，促进人肝细胞的生长，抑制其凋亡，但对非肝脏来源的对照组细胞没有明显影响。
PEG10 gene accelerated the growth of L02 cells and inhibited their apoptosis but it had no conspicuous effect on the non-liver derived cells.
该质粒经过稳定筛选后稳定表达于细胞内，促进人肝细胞的生长，抑制其凋亡，但对非肝脏来源的对照组细胞没有明显影响。
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