The principal pathophysiological mechanism of acute ischemic coronary syndrome(AICS) is atherosclerotic plaque rupture.
急性冠状动脉综合征发生的主要病理生理机制系粥样斑块破裂。
Acute coronary syndromes, including acute cardiac ischemia and sudden coronary death, are frequently caused by a coronary atherosclerotic plaque rupture.
冠状动脉粥样硬化斑块易发斑块破裂，导致急性冠状动脉综合征，引起急性心肌梗死。
The instability of atherosclerotic plaques is the important reason that may result in plaque rupture and then cause stroke.
动脉粥样硬化斑块不稳定是导致斑块破裂进而引起卒中的重要原因。
Arterial thrombi usually form in regions of disturbed flow and at sites of rupture of an atherosclerotic plaque, which exposes the thrombogenic subendothelium to platelets and coagulation proteins;
动脉血栓通常形成于血液湍流区和粥样癍块破裂区，这些部位的内皮下组织暴露于血小板和凝血蛋白。
The instability of atherosclerotic plaques is the leading pathogenesis of stroke resulting in plaque rupture and ulcer-caused thrombosis and it is very harmful.
动脉粥样硬化斑块的不稳定是导致斑块破裂、溃疡致使动脉内血栓形成从而引起卒中的主要发病机制，具有极大的危害性。
Objective: Many study suggested that inflammation plays a crucial role in initiation, evolution and rupture of atherosclerotic plaque of coronary heart disease(CHD)patients.
目的：研究表明炎症在冠心病患者不稳定斑块的发生、演变和斑块破裂过程中起着至关重要的作用。
Objective: Many study suggested that inflammation plays a crucial role in initiation, evolution and rupture of atherosclerotic plaque of coronary heart disease(CHD)patients.
目的：研究表明炎症在冠心病患者不稳定斑块的发生、演变和斑块破裂过程中起着至关重要的作用。
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