Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Objective:To investigate whether brain surface cooling(BSC) can decrease cerebral content of water and Evans Blue(EB) after ischemia-reperfusion injury in experimental rabbits.
目的：探讨脑表面降温对缺血再灌注损伤后的脑组织含水量和伊纹氏蓝含量的影响。
Conclusion DG can markedly inhibit nerve cell apoptosis induced by focal brain ischemia reperfusion injury and protect the nerve cell to a certain extent.
结论DG可明显抑制局灶性脑缺血-再灌注所诱导的脑神经细胞凋亡，起到一定的神经保护作用。
Conclusion: the calcium overload and free radical injury were correlated with brain ischemia reperfusion injury in the aged rats.
结论：老龄大鼠脑缺血再灌注脑组织损伤与钙超载和自由基损伤有关。
Purpose to investigate the anti-inflammatory effect of angiotensin-converting enzyme inhibitor captopril on the brain against ischemia-reperfusion injury and explore its mechanism of action.
目的观察血管紧张素转化酶抑制药卡托普利对大鼠局灶性脑缺血再灌注损伤后炎症反应的影响。
Conclusions the brain ischemia reperfusion injury in young and the aged rats were correlated with the decrease of NO level and the increase of TNF.
结论青年大鼠和老龄大鼠脑缺血再灌注脑组织损伤与NO含量降低和TNF增高有关。
Objective:To study the change of the content of transmitter amino acids of cerebral cortex and hippocampus in mice brain after repetitive ischemia-reperfusion(I/R)injury.
提示脑缺血性损伤与脑组织内递质性氨基酸兴奋抑制失衡有密切关系。
AIM: To investigate the effects of rolipram on the ability of learning and memory and the activity of PDE4 in hippocampus following the focal brain injury induced by ischemia - reperfusion in rats.
目的：观察咯利普兰对局灶性脑缺血-再灌注损伤大鼠学习记忆能力及海马pde4活性的影响。
Objective To investigate the protective effects of propofol on the brain in ischemia-reperfusion (I/R) injury rats and the underlying mechanism.
目的探讨丙泊酚预处理对大鼠全脑缺血再灌注损伤的保护作用。
CONCLUSION It indicated that tea catechins had protective effects on the injury of cerebral ischemia and reperfusion in rats, and these effects may be related to calcium concentrations in brain.
结论茶儿茶素可能通过降低脑细胞内钙离子浓度，保护脑组织对抗缺血再灌注损伤。
CONCLUSION: MT shows significantly protective effect against repeated ischemia reperfusion injury in mice, and the mechanism might be related to inhibit the rise of MDA and NO in brain tissue.
结论：MT对反复脑缺血再灌注损伤有显的保护作用，其机制可能与抑制脑水肿及抑制脑内升高的MDA、NO有关。
Hyperglycemia significantly increases brain damage, mortality rate, and long-term disability after cerebral ischemia and reperfusion injury.
高血糖症会显著增加脑损伤，导致病死率升高，并由脑缺血再灌注损伤后导致患者长期残疾。
Objective To explore the protective of Naloxone hydrochloride on brain and its mechanism during complete brain ischemia-reperfusion injury.
目的探讨纳洛酮对全脑缺血再灌注损伤的防治作用及其机理。
Objective To explore the protective of Naloxone hydrochloride on brain and its mechanism during complete brain ischemia-reperfusion injury.
目的探讨纳洛酮对全脑缺血再灌注损伤的防治作用及其机理。
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