The research about cardiac myocytes renewal is a hot topic in cardiovascular area.
心脏细胞再生的研究是当前心血管研究领域的热点之一。
Precursor cells destined to differentiate into cardiac myocytes (myocytes, cardiac).
是指分化成心肌细胞(肌细胞，心脏)的前体细胞。
Angiotensin increases protein synthesis in cardiac myocytes and DNA synthesis in cardiac fibroblasts.
血管紧张素可提高心肌细胞的蛋白质合成和心肌纤维细胞的DNA合成。
With the use of a specific protocol, these cells were then directed to differentiate into cardiac myocytes.
通过一个特定的程序使这些细胞定向分化为心肌细胞。
Stem cells have the ability to form many fully differentiated cells of the body, including cardiac myocytes.
干细胞具有分化成多种机体组织细胞，包括心肌细胞的潜能。
AIM: To research protective effect of Sanguis Draconis flavones on cell injury in primary cultured cardiac myocytes.
目的：研究龙血竭总黄酮对乳鼠损伤心肌细胞的保护作用。
The study of beating frequencies of cultured cardiac myocytes is an important subject in study of biological rhythms.
离体培养的单个心肌细胞的搏动频率特性的研究是生物节律研究的重要内容之一。
Objective: To study the morphological changes of cardiac myocytes and the subcellular structure of overload hart failure.
目的：研究心衰时心肌细胞及亚细胞结构的形态学改变。
Methods: The transmission electron microscopy was used to study the ultrastructure of the cardiac myocytes in right atrium.
方法取SHR大鼠的右心房作透射电镜观察，分析心肌细胞的超微结构。
FK506 can apparently inhibit the apoptosis of cardiac myocytes, which may be an important way of its immunosuppressive actions.
FK5 0 6能明显抑制心肌细胞凋亡，这可能是FK5 0 6发挥免疫抑制作用的重要方面。
AIM: To characterize the single channel properties of swelling-activated chloride channel in freshly isolated mouse cardiac myocytes.
目的研究急性分离小鼠心肌细胞肿胀激活氯离子通道的单通道特征。
By centrifugal selection, differential paces of sticking to wall and medical inhibit method, purification of cardiac myocytes reach to 95 %.
进一步通过离心、差速贴壁和化学试剂抑制非心肌细胞生长，纯化得到95%以上的心肌细胞。
AIM: To study the effect of insulin on proliferation and hypertrophy of cardiac myocytes and its role in the induction of cardiac hypertrophy.
目的：研究胰岛素对心脏细胞增殖和心肌细胞肥大的影响，探讨其在心肌肥厚中的作用。
Physiological growth of normal cardiac myocytes was due to increase in cell volume, i. e. the cell length and cross-section area were enlarged.
正常心肌细胞的生长，是由于细胞的长度和横切面积的增加，从而使心肌细胞体积增大。
The results provide a new method to study the selective innervation of cardiac myocytes and the interactions between neurons and cardiac myocytes.
这一结果对于进一步研究心肌细胞的选择性神经支配以及神经与心肌的相互作用开辟了一条新的研究途径。
The increase of circulating catecholamines exerts direct toxic effect on cardiac myocytes, increases membrane permeability and myocardial fibrosis;
循环儿茶酚胺升高对心肌细胞产生直接的毒性作 用、增加膜通透性和心肌纤维化，这些作用可引起细胞死亡。
Results The levels of LPL mRNA in cardiac myocytes of diabetic group increased, but though BMOV treatment the levels of LPL mRNA decreased to control levels.
结果糖尿病大鼠心肌脂蛋白脂酶mRNA是升高的，经BMOV治疗后心肌细胞脂蛋白脂酶m RNA表达可降低，基本恢复到正常对照水平。
A: Adrenaline from the nervous system lands on receptors of cardiac myocytes (heart-muscle cells), and this causes calcium channels in the membranes of those cells to open.
答：来自神经系统的肾上腺激素“着陆”到心脏肌细胞的受体，这使得这些肌细胞的膜中的钙通道打开。
Gap junctions are the direct pathways to exchange signals among cardiac myocytes. The electrical conduction velocity there is faster than other place on cell membrane.
缝隙连接是细胞间信息交换的直接通道，此处的电传导速度比细胞膜上其他部位都快。
They provide structural support for cardiac myocytes and are responsible for extracellular matrix synthesis in the heart during growth and pathophysiological conditions.
它们对于心肌细胞起结构支持作用，在生长和病理条件下，负责细胞外基质的合成。
AIM: to investigate the inductive effects of D-galactose, which can induce cell aging of fibroblasts and neurons, on the modeling senescence of cardiac myocytes in rats.
目的：观察可诱导成纤维细胞、神经元细胞衰老的D -半乳糖对心肌细胞拟衰老的诱导作用。
Methods: Actin in cardiac myocytes of rats with chronic heart failure induced by banding of ascending aorta was analysed qualitatively and hemi-quantitatively via SP method.
方法：采用SP免疫组化方法，对升主动脉缩窄慢性心衰大鼠心肌细胞肌动蛋白进行定性及半定量分析。
Another strength is the use of stereologically correct sampling and sectioning procedures, which allows estimation of the total number of cardiac myocytes in each individual LV.
使用立体学方法校正样本和切片操作以评估每个左心室心肌细胞的总数量。
Conclusion Simvastatin can inhibit the hypertrophy of cultured rat cardiac myocytes induced by serum, the mechanism of which might be related with the increase of PTEN expression.
结论辛伐他汀能够抑制血清诱导的心肌细胞肥大，升高pten表达水平可能是其分子生物学机制之一。
Sarcolemmal membrane permeability alteration of cardiac myocytes in rats fed on grains from Keshan disease endemic area was studied with electron microscopy, using La(NO3)3(La3 + )as the tracer.
以硝酸镧作示踪物用电子显微镜观察喂饲克山病病区粮3个月的大白鼠心肌细胞膜通透性的变化。
Myofibrillar fragmentation, swelling of the cardiac myocytes, absence of the Z line, increases of the mitochondria and collagen fibers were found in ouabain group by transmission electron microscopy.
透射电镜观察显示，哇巴因组大鼠心肌细胞肿胀，肌纤维断裂，Z线消失，线粒体增生肿胀，部分空泡化，细胞间胶原纤维增生。
Results Type I collagen was distributed in myocytes, and it encased each cardiac cell. The densities of collagen network in difference cardiac part were of disparity.
结果I型胶原分布广泛，包裹大鼠心每一个心肌细胞，根据部位的不同，胶原网络的密疏程度有差异。
And we have done that now and the fibroblasts now express the cardiac genes involved in the development of myocytes.
我们目前已经将该步骤完成，成纤维细胞现在已经表达了参与心肌细胞形成的心脏基因。
And we have done that now and the fibroblasts now express the cardiac genes involved in the development of myocytes.
我们目前已经将该步骤完成，成纤维细胞现在已经表达了参与心肌细胞形成的心脏基因。
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