Survival duration of mice after cerebral anoxia.
脑缺氧后小鼠存活时间。
Results The main cause of acute encephalocele were delayed intracranial hematomas, acute diffuse brain swelling, cerebral contusion and laceration in Sylvain tissue and ischemic anoxia.
结果迟发性颅内血肿、弥漫性脑肿胀、侧裂区脑挫裂伤、脑组织缺血、缺氧等是重型颅脑损伤术中急性脑膨出的主要原因。
BACKGROUND: After cerebral tissue ischemia and anoxia in young rats, the cerebral edema gets serious, and the levels of nitric oxide (no) and malondialdehyde (MDA) decrease.
背景：幼鼠脑缺氧缺血后，脑组织水肿加重，脑组织中一氧化氮及丙二醛水平增高。
Objective To observe the dynamic change of nitric oxide (no) contents in the cerebral tissues of mices of anoxia asphyxia.
目的研究小鼠缺氧窒息后脑组织一氧化氮(NO)含量的动态变化。
Objective:To study the effects of grape seed proanthocyanidins(GSP) on injuries of cerebral ischemia and reperfusion and anoxia in mice.
目的：研究葡萄籽原花青素（GSP）对小鼠脑缺血、再灌注损伤及常压缺氧的影响。
Methods (1) The experiments of quick decapitation and anoxia under normal pressure model in mice was used to study the effects of LW on acute cerebral ischemia and anoxia.
方法：（1）采用小鼠断头实验和常压耐缺氧实验，观察LW对脑组织急性缺血缺氧的保护作用；
MethodsThe general level experiment was applied acute cerebral ischemia or anoxia experimental method.
整体水平实验部分采用急性脑缺血缺氧的实验方法。
Results: Extract liquid Qishengwan could obviously improve the protective effect on the mice anoxia, revive the SOD activity of the cerebral tiss.
目的：观察七圣丸水提物抗缺氧与抗脂质过氧化作用。
These results indicated that squalene could protect mice from anoxia caused by acute cerebral ischemia and sodium nitrite poisoning.
表明角鲨烯对急性脑缺血性和亚硝酸钠中毒缺氧具有保护作用。
These results indicated that squalene could protect mice from anoxia caused by acute cerebral ischemia and sodium nitrite poisoning.
表明角鲨烯对急性脑缺血性和亚硝酸钠中毒缺氧具有保护作用。
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