Ischemia could stimulate neurogenesis in the cerebral cortex and hippocampus which is considered as a very important neuronal protection mechanism.
脑缺血可上调大脑皮层和海马的神经元再生，这可能是一种重要的神经保护机制。
Objective To investigate the protection of brief focal cerebral ischemic preconditioning against microvascular injury and brain edema after subsequent severe focal ischemia.
目的研究短暂局灶性脑缺血预处理对再次脑缺血后微血管损伤及脑水肿的保护作用。
CONCLUSION: the increase of neuron EGFR expression can be induced by focal cerebral ischemia injury, and may be related to the self protection of ischemia neurons.
结论：局灶性脑缺血损伤可诱导神经元egfr表达增加，EGFR表达增加可能与缺血神经元自身保护作用有关。
Conclusion: the high expressions of HGF in rat brain might play an important role in the protection and repair of the damaged tissue induced by focal cerebral ischemia.
结论：HGF在局灶脑缺血诱导的高表达可能在脑缺血损伤组织的保护及修复过程中起重要作用。
Objective To observe the effect of mild hypothermia on cerebral protection during temporarily blocking partent arteries of intracranial giant aneurysms and ischemia-reperfusion period.
目的观察亚低温对颅内巨大动脉瘤夹闭手术中临时阻断动脉血流及缺血-再灌注期的脑保护作用。
Objective To investigate the effects of of TERT in the mechanism (s) of protection elicited by electrical stimulation cerebellar fastigial nucleus before focal cerebral ischemia reperfusion in rats.
目的探讨tert在预电刺激小脑顶核后脑缺血再灌注保护机制中的作用及可能的作用途径。
OMT has protection against injury induced by cerebral ischemia in rats. The anti-inflammation effects may be one of the mechanisms.
氧化苦参碱对局灶性脑缺血大鼠的脑缺血性损伤具有明显的保护作用，抗炎可能为其作用途径之一。
Conclusion Resveratrol can induce expression of VEGF in early phase after cerebral ischemia, which promotes the formation of new blood vessels in ischemic area playing a role in cerebral protection.
结论白藜芦醇可以诱导脑缺血后vegf早期表达，促进缺血区域新生血管形成，发挥脑保护作用。
Conclusion OMT has protection against injury induced by cerebral ischemia in rats. The anti-inflammation effects may be one of the mechanisms.
结论：氧化苦参碱对局灶性脑缺血大鼠的脑缺血性损伤具有明显的保护作用，抗炎可能为其作用途径之一。
Conclusion OMT has protection against injury induced by cerebral ischemia in rats. The anti-inflammation effects may be one of the mechanisms.
结论：氧化苦参碱对局灶性脑缺血大鼠的脑缺血性损伤具有明显的保护作用，抗炎可能为其作用途径之一。
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