Objective To explore the role of uncoupling proteins (UCP) in resisting diet-induced obesity in rats.
目的探讨解偶联蛋白在大鼠抵抗饮食诱导肥胖中的作用。
Objective: To explore the establishment of diet-induced obesity model in pubertal male rats with high fat diet.
目的：以高脂饲料配方建立青春期单纯性肥胖雄性大鼠模型。
Objective In order to explore the role of orexin A, B in appetite regulation of diet-induced obesity resistant (DIO-R) rats.
目的探讨增食因子A和B在肥胖抵抗大鼠食欲调节中的作用。
Objective To study the change of obesity related index in diet-induced obesity-resistant (DIO-R) rats and diet-induced obesity (DIO) rats.
目的探讨饮食诱导肥胖抵抗(DIO - R)大鼠和饮食诱导肥胖(DIO)大鼠的肥胖相关指标的变化。
There was difference of fat rich diet induced obesity and insulin resistance in the same species rats, the obesity prone rats had more tendency to become insulin resistance.
同一种系大鼠中存在饮食诱导肥胖和胰岛素抵抗的个体差异，肥胖抵抗大鼠不易发生胰岛素抵抗。
There was difference of fat - rich - diet induced obesity and insulin resistance in the same species species rats, the obesity - prone rats had more tendency to become insulin resistance.
在同一种系大鼠中存在饮食诱导肥胖和胰岛素抵抗的个体差异，肥胖抵抗大鼠不易发生胰岛素抵抗。
Objective To explore the gene expression of hormone sensitive lipase (HSL) and lipoprotein lipase (LPL) between obesity-prone (op) and obesity-resistant (or) rats induced by a high-fat diet.
目的探讨高脂饮食诱导肥胖易感(OP)大鼠和肥胖抵抗(OR)大鼠，激素敏感性脂肪酶(HSL)和脂蛋白脂肪酶(LPL)基因表达的差别。
At the end of the experiment, the high-fat diet group was divided into dietary induced obesity (DIO) and dietary induced obesity resistant (DIO-R) subgroups according to the final body weight.
实验结束时，根据体重将高脂实验组大鼠分为饮食诱导肥胖(DIO)和饮食诱导肥胖抵抗(DIO R)大鼠，比较各组相关指标的差异。
Objective: To assess the anti-obesity effect and mechanism of Jianzhi capsules on the obese rats induced by high fat diet.
目的：研究中药复方制剂减之胶囊对肥胖大鼠的减肥降脂作用及其作用机制。
Conclusions: To make the model of high-fat diet induced obesity-prone and obesity-resistant rats is according with the biochemistry characteristic of high-fat diet in public.
结论：高脂饮食诱导的肥胖易感和肥胖抗性大鼠模型制备成功，其生化特征符合人群高脂饮食致肥胖的特点。
Conclusions: To make the model of high-fat diet induced obesity-prone and obesity-resistant rats is according with the biochemistry characteristic of high-fat diet in public.
结论：高脂饮食诱导的肥胖易感和肥胖抗性大鼠模型制备成功，其生化特征符合人群高脂饮食致肥胖的特点。
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