Conclusion GLAST rather than GLT-1 may play a crucial role in excitotoxicity induced by rotenone.
结论谷氨酸转运体GLAST可能在鱼藤酮诱导的兴奋性损伤机制中起主要作用。
In pathological conditions, zinc effect also contributes to the excitotoxicity-induced neuronal death.
病理条件下，锌离子参与了兴奋性细胞毒作用所触发的神经元凋亡过程。
In pathological conditions, zinc effect also contributes to the excitotoxicity-induced neuronal death.
病理条件下，锌离子参与了兴奋性细胞毒作用所触发的神经元凋亡过程。
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