DATA SYNTHESIS: During continuous exercise, myocardium hypertrophy induced by exercise was the physical basis of the increase in cardiac output.
资料综合：在持续性运动期间运动诱导心肌肥大是心输出量增加的生理基础。
Objective:To investigate the changes of plasma biological active peptides in exercise-induced cardiac hypertrophy in rats.
目的：研究运动性心肌肥大形成中血浆生物活性肽的含量及意义。
In this article we only reviewed the biological mechanism of exercise induced cardiac hypertrophy by its inductive stimulus, pass way for signal transduction and genetic expression.
仅从运动性心肌肥大的生物学机制方面，就诱导其发生的刺激因素及其信号转导通路、基因表达等几个方面加以综述。
It suggested that the ability of anti-injury in exercise-induced cardiac hypertrophy is higher than that in hypertension.
本研究提示运动性心肌肥大的心肌抗缺血再灌注损伤的能力较高血压心肌肥大者增强。
Conclusion QTd was decreased in exercise-induced cardiac hypertrophy subjects, which may reflect homogeneous myocardial repolarization in long term trained subjects.
结论运动诱导的心肌肥厚使QT离散度减小，反映了长期运动个体心肌复极化的稳定性。
Conclusion QTd was decreased in exercise-induced cardiac hypertrophy subjects, which may reflect homogeneous myocardial repolarization in long term trained subjects.
结论运动诱导的心肌肥厚使QT离散度减小，反映了长期运动个体心肌复极化的稳定性。
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