It is important to reduce ischemia reperfusion injury.
重要的是要降低缺血再灌注损伤。
INTERVENTIONS: Rat complete cerebral ischemia reperfusion model was established.
干预：制备大鼠全脑缺血再灌注模型。
Methods: Rabbit lung model of ischemia reperfusion injury was established in vivo.
方法：采用在体兔单肺原位缺血再灌注损伤模型。
Conclusion: Wenxin soup can protect myocardial injury of ischemia reperfusion by IP way.
结论：温心汤通过预处理途径对心肌缺血再灌注损伤产生保护作用。
Objective: Study the influence on rat brain edema and ATPase activity with cerebral ischemia reperfusion.
目的：研究缺血再灌注对脑组织水肿和ATP酶活性的影响。
Aim: to evaluate the effect of rhubarb on hepatocyte injury after intestinal ischemia reperfusion in rats.
目的：评价大黄对大鼠肠缺血所致肝细胞损伤的防治作用。
Objective To observe the protective effects of flunarizine on ischemia reperfusion injury in rabbit retina.
目的动态观察盐酸氟桂嗪对家兔视网膜缺血-再灌注损伤的保护作用。
Objective To investigate the effect of Shenlibao against myocardial ischemia reperfusion injury in rabbits.
目的观察参力保对家兔心肌缺血再灌注损伤的保护作用。
Objective To clarify the effect of nitrous oxide for the treatment of spinal cord ischemia reperfusion injury.
目的研究一氧化氮在脊髓缺血再灌注损伤中的作用。
Objective To evaluate the protective effect of scopolamine against acute cerebral ischemia reperfusion injury.
目的探讨东莨菪碱对急性脑缺血再灌注损伤的保护作用。
Objective To study the protection of ischemic preconditioning (IPC) on the hepatic ischemia reperfusion injury.
目的评价缺血预处理（IPC）对大鼠肝脏的保护作用。
This review was performed to summarize the possible mechanism and therapy in ischemia reperfusion injury of intestine.
本文概述了肠缺血再灌注损伤的可能机理及其预防和治疗措施。
The present study strongly indicate that batroxobin inhibits apoptosis induced by cerebral ischemia reperfusion injury.
本实验提示巴曲酶对脑缺血再灌流损伤所引起的细胞凋亡有抑制作用。
Methods Cerebral ischemia reperfusion injury model was produced in rats. The electrolyte contents were measured with atomic absorption spectrophotometer(AAS).
方法建立大鼠急性脑缺血再灌流损伤模型，用原子分光光度仪检测脑组织电解质含量。
Objective: to explore research the protective effect of ischemia preconditioning to rabbits 'myocardium injury of ischemia reperfusion, and discuss its mechanism.
目的：从细胞保护角度出发，研究缺血预适应对家兔心肌缺血再灌注损伤的保护作用，并探讨其机制。
Conclusion Inhibiting activation of Kupffer cells may attenuate hepatic ischemia reperfusion injury, and Kupffer cells may play an important role in hepatic ischemia reperfusion injury.
结论抑制枯否细胞活化可减轻肝脏缺血再灌注损伤，枯否细胞在肝脏缺血再灌注损伤中的作用很重要。
Conclusion PKC took place translocation activation during cerebral ischemia reperfusion, PKC promoted the expression of FOS and BCL 2. Their complex role influenced the neuronal apoptosis.
结论缺血再灌注期间PKC发生易位激活，PK C促进了FOS和BCL -2的表达，这几个因素综合作用影响神经细胞凋亡。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Objective To observe the protective effect of ischemic preadaptation on myocardium injury of ischemia reperfusion by adopting the animal model of ischemia reperfusion on myocardium injury rabbits.
目的采用家兔心肌缺血再灌注损伤动物模型，观察缺血预适应对心肌缺血再灌注损伤的心肌保护作用。
Conclusion: Yufeng Capsule could prevent and treat the cerebral edema caused by ischemia reperfusion, its mechanism may be related to improving cytomembrane ionic transport after ischemia reperfusion.
结论：御风胶囊能防治缺血再灌注所致脑水肿，其机理可能与其改善脑缺血再灌注后细胞膜离子转运有关。
Objective: To study the effects of L-carnitine on myocardial ischemia-reperfusion injury and energy metabolism in rats.
目的：观察左旋卡尼汀对离体大鼠心脏缺血-再灌注损伤的作用及对心肌细胞能量代谢的影响。
Ischemic preconditioning 24 hours before ischemia-reperfusion injury increased VEGF-A levels and substantially decreased the number of apoptotic retinal cells.
在缺血-再灌注损伤前24小时进行缺血的预处理可增加VEGF - A水平并大量减少凋亡的视网膜细胞数量。
Objective To study the protective effect of hyperoxic liquid preconditioning in myocardial ischemia-reperfusion injury.
目的探讨高氧液预处理对兔心肌缺血再灌注损伤的保护作用。
Objective To observe the changes of platelet count, function and its membrane protein in rabbits undergoing liver ischemia-reperfusion.
目的观察兔肝缺血再灌注过程中血小板数量、功能及膜蛋白的变化。
These results showed that the energy metabolism dysfunction was the initial stage, and the damage of oxygen free radicals was the major factor in ischemia-reperfusion injury.
这些结果说明线粒体能量代谢障碍是缺血再灌注损伤的始动环节，而氧自由基是造成损伤的主要因素。
Objective To investigate the effects of high thoracic epidural anesthesia (HTEA) on cerebral ischemia-reperfusion (I/R) injury, and the possible mechanism.
目的探讨上胸段硬膜外阻滞（HTEA）对大鼠脑缺血再灌注损伤的作用及可能机制。
Objective: To observe the protective effect of tea saponin on Focal cerebral ischemia-reperfusion injury.
目的观察茶皂素对大鼠局灶性脑缺血再灌注损伤的保护作用。
Objective:To investigate whether brain surface cooling(BSC) can decrease cerebral content of water and Evans Blue(EB) after ischemia-reperfusion injury in experimental rabbits.
目的：探讨脑表面降温对缺血再灌注损伤后的脑组织含水量和伊纹氏蓝含量的影响。
Methods: a model of acute ischemia-reperfusion injury was produced in open chest anaesthetized rabbits.
方法：采用麻醉开胸兔急性心肌缺血再灌注损伤模型。
Methods: a model of acute ischemia-reperfusion injury was produced in open chest anaesthetized rabbits.
方法：采用麻醉开胸兔急性心肌缺血再灌注损伤模型。
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