It is important to reduce ischemia reperfusion injury.
重要的是要降低缺血再灌注损伤。
Methods: Rabbit lung model of ischemia reperfusion injury was established in vivo.
方法：采用在体兔单肺原位缺血再灌注损伤模型。
Objective To investigate the effect of Shenlibao against myocardial ischemia reperfusion injury in rabbits.
目的观察参力保对家兔心肌缺血再灌注损伤的保护作用。
Objective To observe the protective effects of flunarizine on ischemia reperfusion injury in rabbit retina.
目的动态观察盐酸氟桂嗪对家兔视网膜缺血-再灌注损伤的保护作用。
Conclusions The apigenin have a prominent protective effect on hepatic ischemia reperfusion injury in rats.
结论芹菜素对大鼠肝脏缺血再灌注损伤有一定的保护作用。
Objective To clarify the effect of nitrous oxide for the treatment of spinal cord ischemia reperfusion injury.
目的研究一氧化氮在脊髓缺血再灌注损伤中的作用。
Objective To evaluate the protective effect of scopolamine against acute cerebral ischemia reperfusion injury.
目的探讨东莨菪碱对急性脑缺血再灌注损伤的保护作用。
Objective To study the protection of ischemic preconditioning (IPC) on the hepatic ischemia reperfusion injury.
目的评价缺血预处理（IPC）对大鼠肝脏的保护作用。
Objective To investigate the protective effects of ginsenosides (GS) on renal ischemia reperfusion injury in rats.
目的观察研究人参总皂甙(GS)对肾缺血再灌注损伤的保护作用。
This review was performed to summarize the possible mechanism and therapy in ischemia reperfusion injury of intestine.
本文概述了肠缺血再灌注损伤的可能机理及其预防和治疗措施。
The present study strongly indicate that batroxobin inhibits apoptosis induced by cerebral ischemia reperfusion injury.
本实验提示巴曲酶对脑缺血再灌流损伤所引起的细胞凋亡有抑制作用。
Conclusions Ginseng-monkshood extract injection has obviously protective effect on myocardial ischemia reperfusion injury.
结论参附注射液对缺血再灌注损伤心脏具有保护作用。
Conclusion: the calcium overload and free radical injury were correlated with brain ischemia reperfusion injury in the aged rats.
结论：老龄大鼠脑缺血再灌注脑组织损伤与钙超载和自由基损伤有关。
OBJECTIVE: to study the influence of different doses of naloxone on focal cerebral ischemia reperfusion injury in rats and to explore its mechanism.
目的：观察不同剂量的纳洛酮对大鼠局灶性脑缺血再灌注损伤模型的影响，并探讨其保护作用机制。
Methods Cerebral ischemia reperfusion injury model was produced in rats. The electrolyte contents were measured with atomic absorption spectrophotometer(AAS).
方法建立大鼠急性脑缺血再灌流损伤模型，用原子分光光度仪检测脑组织电解质含量。
The application of calcium antagonists during ischemia reperfusion injury can inhibit the inward flow of calcium ions and decrease the severity of histiocytic destruction.
应用钙离子拮抗剂在组织缺血再灌注损伤过程中可以抑制钙离子内流，降低组织细胞的破坏程度。
Conclusion Inhibiting activation of Kupffer cells may attenuate hepatic ischemia reperfusion injury, and Kupffer cells may play an important role in hepatic ischemia reperfusion injury.
结论抑制枯否细胞活化可减轻肝脏缺血再灌注损伤，枯否细胞在肝脏缺血再灌注损伤中的作用很重要。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Conclusion Nitric oxide could increase the release of the hepatic enzymes after liver ischemia reperfusion injury, inhibit the apoptosis of hepatocytes and improve liver ischemia reperfusion injury.
结论一氧化氮可减少肝缺血再灌注损伤后肝酶的释放，抑制肝细胞的凋亡，改善肝缺血再灌注损伤。
Objective: To observe the protective effect of tea saponin on Focal cerebral ischemia-reperfusion injury.
目的观察茶皂素对大鼠局灶性脑缺血再灌注损伤的保护作用。
Objective To study the protective effect of hyperoxic liquid preconditioning in myocardial ischemia-reperfusion injury.
目的探讨高氧液预处理对兔心肌缺血再灌注损伤的保护作用。
Objective:To investigate whether brain surface cooling(BSC) can decrease cerebral content of water and Evans Blue(EB) after ischemia-reperfusion injury in experimental rabbits.
目的：探讨脑表面降温对缺血再灌注损伤后的脑组织含水量和伊纹氏蓝含量的影响。
Methods: a model of acute ischemia-reperfusion injury was produced in open chest anaesthetized rabbits.
方法：采用麻醉开胸兔急性心肌缺血再灌注损伤模型。
Conclusion: CVB-D has protective effect on cerebral ischemia-reperfusion injury in rats.
结论：CVB - D对大鼠局灶性脑缺血再灌注损伤具有保护作用。
Objective: To study the effects of L-carnitine on myocardial ischemia-reperfusion injury and energy metabolism in rats.
目的：观察左旋卡尼汀对离体大鼠心脏缺血-再灌注损伤的作用及对心肌细胞能量代谢的影响。
Ischemic preconditioning 24 hours before ischemia-reperfusion injury increased VEGF-A levels and substantially decreased the number of apoptotic retinal cells.
在缺血-再灌注损伤前24小时进行缺血的预处理可增加VEGF - A水平并大量减少凋亡的视网膜细胞数量。
These results showed that the energy metabolism dysfunction was the initial stage, and the damage of oxygen free radicals was the major factor in ischemia-reperfusion injury.
这些结果说明线粒体能量代谢障碍是缺血再灌注损伤的始动环节，而氧自由基是造成损伤的主要因素。
Objective To evaluate the effect of ligustrazine and ulinastatin on protecting ischemia-reperfusion injury after limb replantation.
目的：评价川芎嗪和乌司他丁对断肢再植缺血再灌注损伤的保护作用。
Objective To evaluate the effect of ligustrazine and ulinastatin on protecting ischemia-reperfusion injury after limb replantation.
目的：评价川芎嗪和乌司他丁对断肢再植缺血再灌注损伤的保护作用。
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