CONCLUSION: GLUT1 expression is notably up-regulated in the penumbra region after focal cerebral ischemia, it may be a protective reaction against ischemic injury.
结论：GLUT1在缺血半影区的表达上调，可能是机体对缺血损伤的保护性反应。
The clinical treatment of the key is to save the ischemic region (penumbra) of dying neurons and promote damage nerve function recovery.
临床上治疗的关键在于挽救缺血区域（半暗带）的濒临死亡的神经元和促进损伤后神经功能的恢复。
To recover blood supply of brain tissue in ischemic region especially in ischemic penumbra in time, it will have great significance in treating and prognosis.
脑缺血后，及时恢复缺血区脑组织的血液供应，尤其是缺血半暗带区的血液供应，对于缺血性脑血管病的治疗与预后意义重大。
To recover blood supply of brain tissue in ischemic region especially in ischemic penumbra in time, it will have great significance in treating and prognosis.
脑缺血后，及时恢复缺血区脑组织的血液供应，尤其是缺血半暗带区的血液供应，对于缺血性脑血管病的治疗与预后意义重大。
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