Protective effect of panax notoginsenosides on renal ischemia reperfusion injury in rats;
目的观察三七总苷对大鼠肾缺血再灌注损伤的保护作用。
Objective To investigate the protective effects of ginsenosides (GS) on renal ischemia reperfusion injury in rats.
目的观察研究人参总皂甙(GS)对肾缺血再灌注损伤的保护作用。
To investigate the expression of intercellular adhesive molecular 1 (ICAM 1) in renal ischemia reperfusion injury.
观察肾缺血再灌注过程中，细胞间粘附分子1 (ICAM 1)在肾组织中的表达。
Conclusions: tea polyphenols preconditioning group to kidney could protect the kidney against renal ischemia reperfusion injury.
结论：在肾脏缺血再灌注损伤过程中，茶多酚能起到对肾脏的保护作用。
Objective To investigate the effect of Astragalus on expression of intercellular adhesive molecule 1 (ICAM 1) in renal ischemia reperfusion injury.
目的探讨黄芪在肾缺血再灌注损伤中对细胞粘附分子1（ICAM 1）表达的影响。
Objective: to observe the influence of Astragalus injection on the apoptosis of cells in rats after renal ischemia reperfusion injury in order to elucidate the mechanism of its protective effect.
目的：观察黄芪注射液对大鼠肾脏缺血再灌注细胞凋亡的影响，探讨其对肾脏缺血再灌注损伤的保护机制。
Renal injury after ischemia appears to be a consequence of tissue hypoxia not only from interrupted blood supply but also from the process of reperfusion which leads to an active inflammatory process.
肾缺血后损伤显示，损伤不仅是血供中断后组织缺氧的结果，更有再灌注进程中引起炎症反应的激活过程。
Purpose: To investigate the role of heat shock protein 70 (HSP70) in renal ischemia-reperfusion injury in rats after ischemic preconditioning by examining the change of the expression of HSP70.
目的：研究缺血预处理对肾脏缺血再灌注损伤的保护作用，观察热休克蛋白70 (HSP70)的表达变化并探讨其作用机制。
Objective to explore the effects of hypertonic saline preconditioning on the expression of intercellular adhesion molecules-1 (ICAM1) after renal ischemia-reperfusion injury in rats.
目的探讨高渗盐液预处理对大鼠肾缺血再灌注损伤后肾组织细胞间黏附分子- 1的表达的影响。
Conclusion PROG may exacerbate the renal injury of ischemia-reperfusion.
结论黄体酮可能加剧肾缺血-再灌注损伤。
These results suggest that oxygen free radicals may damage the activity of cytochrome oxidase, the reduction of which may play an important role in the injury of renal ischemia and reperfusion.
提示氧自由基可能损害细胞色素氧化酶活性，细胞色素氧化酶活性降低在肾缺血再灌流损伤中可能起重要作用。
Objective To evaluate the functional alternations of canine renal ischemia and reperfusion injury by using MR perfusion-weighted imaging, and to correlate the imaging with the pathologic findings.
目的结合病理对照研究MR灌注成像评价犬肾缺血再灌注损伤后肾功能的变化。
The mechanism of the acute tubular necrosis (ATN) caused by acute renal ischemia-reperfusion injury is complicated and there are many correlation factors in it.
急性缺血再灌注引起急性肾小管坏死发生机制较为复杂，相关因素比较多。
Animal ARF can be induced by renal ischemia-reperfusion injury, glycerine, gentamicin and mercuric chloride, and typical acute tubular necrosis can be observed in pathological changes.
肾动脉钳闭引起的肾脏缺血再灌注以及甘油、庆大霉素、氯化汞均可致实验动物急性肾衰竭，病理学上可见典型的急性肾小管坏死。
Animal ARF can be induced by renal ischemia-reperfusion injury, glycerine, gentamicin and mercuric chloride, and typical acute tubular necrosis can be observed in pathological changes.
肾动脉钳闭引起的肾脏缺血再灌注以及甘油、庆大霉素、氯化汞均可致实验动物急性肾衰竭，病理学上可见典型的急性肾小管坏死。
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