AIM: To introduce and evaluate a canine model of secondary hypersplenism induced by splenic vein ligation.
目的：介绍并评价脾静脉结扎诱导的继发性脾功能亢进犬动物模型。
A canine model of secondary hypersplenism was established by ordinal ligation of gastrosplenic vein trunk, splenic vein and its collateral branches.
通过依次结扎犬脾门处脾静脉主干、胃脾静脉干及脾静脉属枝，建立继发性脾功能亢进动物模型。
Conclusion The method of splenic vein ligation to induce experimental secondary hypersplenism is a simple, effective and relatively ideal model for surgical or interventional therapy in hypersplenism.
结论脾静脉结扎建立继发性功能亢进方法简单、确切，可以作为脾功能亢进外科或介入治疗的较理想模型。
In group B, 8 patients were found to suffer from hypersplenism secondary to liver cirrhosis.
组为8例肝炎后性肝硬化继发脾机能亢进病人。
Methods 20 cases underwent splenectomy, including 4 cases of hypersplenism secondary to hepatic cirrhosis, 1 case of pseudocyst of spleen, 14 cases of primary thrombocytopenic purpura.
方法需行脾切除术的患者2 0例，包括肝硬化继发性脾亢4例，脾假性囊肿1例，遗传性球形细胞增多症1例，原发性血小板减少性紫癜14例。
Methods 20 cases underwent splenectomy, including 4 cases of hypersplenism secondary to hepatic cirrhosis, 1 case of pseudocyst of spleen, 14 cases of primary thrombocytopenic purpura.
方法需行脾切除术的患者2 0例，包括肝硬化继发性脾亢4例，脾假性囊肿1例，遗传性球形细胞增多症1例，原发性血小板减少性紫癜14例。
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