Will the therapy for renal osteodystrophy exacerbate vascular calcification?
对肾性骨病的治疗会加剧血管钙化吗？
The poor response to vasopressor might be due to excessive oxidative stress and vascular calcification.
而对升压药反应不良的原因可能是过度氧化压力及血管钙化所导致。
Prevention of vascular calcification with bisphosphonates without affecting bone mineralization: a new challenge?
不影响骨质矿化的二膦酸盐预防血管钙化的作用：新的挑战？
The possible correlation of vascular calcification with BMP-2 and BMP-7 is attracting the most attention at present.
骨形成蛋白2和骨形成蛋白7与血管钙化的可能相关作用是目前研究最多的。
Furthermore, to determine the effect of Fluvastatin on vascular calcification in vitro and its possible mechanism. Methods.
此外，加用氟伐他汀进行干预，观察其对体外血管钙化的影响并探讨可能的作用机制。
Conclusions Vascular calcification is related to the deposition of the bone matrix proteins in the inferior epigastric artery of ESRD patients.
结论尿毒症患者腹壁下动脉的钙化与骨特异性蛋白的表达有关。
Objective to explore pathological features of peripheral artery calcification in uremic patients and relationship between vascular calcification and clinical risk factors.
目的研究尿毒症患者外周血管钙化的特点，并探讨血管钙化与临床相关因素之间的关系。
Conclusion Our results indicated that ghrelin can significantly attenuate the calcification in aorta and VSMC partly through the antagonism with endothelin system in circulation and vascular tissues.
结果提示，生长素可能部分通过拮抗血浆和血管组织局部的内皮素系统效应而产生抑制血管组织和血管平滑肌细胞钙化的作用。
Relationships between vasculature and viscera can be demonstrated as well as vascular patency, thrombus, or calcification.
同血管的开放度、血栓或钙化一样，脉管系统和内脏之间的关系可以清楚显示出来。
Vascular proliferation, angiectasis, perivascular lymphocyte infiltration, hemorrhage, necrosis, cystoid degeneration and calcification were seen frequently.
血管增生扩张、血管周围淋巴细胞浸润、出血、坏死、囊性变常见。
Vascular proliferation, angiectasis, perivascular lymphocyte infiltration, hemorrhage, necrosis, cystoid degeneration and calcification were seen frequently.
血管增生扩张、血管周围淋巴细胞浸润、出血、坏死、囊性变常见。
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