目的探讨一氧化氮（NO）在缺血预处理（IP）保护大鼠移植肝脏缺血再灌注损伤中的作用。
Objective To investigate the protective effect of nitric oxide (NO) induced by ischemic preconditioning (IP) on ischemic reperfusion injury of rat liver graft.
在缺血-再灌注损伤前24小时进行缺血的预处理可增加VEGF - A水平并大量减少凋亡的视网膜细胞数量。
Ischemic preconditioning 24 hours before ischemia-reperfusion injury increased VEGF-A levels and substantially decreased the number of apoptotic retinal cells.
目的：研究内源性一氧化碳（CO）在大鼠肝脏缺血预处理中的保护作用机制。
Objective:To ascertain whether CO/cGMP pathway is invovled in the protective mechanism of Endogenous carbon monoxide in the rat liver undergone ischemic preconditioning.
结论高氧液预处理对家兔在体心肌缺血再灌注损伤具有明显的保护作用。
Conclusion Hyperoxic liquid preconditioning can protect rabbit myocardial injury caused by ischemic reperfusion.
目的探讨降钙素基因相关肽(CGRP)在临床肝脏缺血预处理中的保护作用。
Objective to study the protective effect of calcitonin gene related peptide (CGRP) in hepatic ischemic preconditioning.
目的探讨CD8+CD28-T细胞在远程缺血预处理保护早期肾脏热缺血再灌注损伤中的作用。
To study the effect and mechanism of CD8+CD28-T cell on remote ischemic preconditioning protects the rat kidney from early warm ischemia-reperfusion injury.
目的探讨CD8+CD28-T细胞在远程缺血预处理保护早期肾脏热缺血再灌注损伤中的作用。
To study the effect and mechanism of CD8+CD28-T cell on remote ischemic preconditioning protects the rat kidney from early warm ischemia-reperfusion injury.
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