气道高反应性通过直接气道阻力分析监测。
Airway hyperresponsiveness was monitored by direct airway resistance analysis.
由哮喘病史，即使目前没有症状，气道高反应性也会发生。
Airway hyperreactivity may occur even in patients with a history of bronchial asthma who are currently asymptomatic.
本文探讨辨病与辨证相结合治疗气道高反应性咳嗽的方法。
Therapies for airway hyper-reactive cough by differentiation of its symptoms and signs were studied.
气道炎症细胞积聚、气道慢性炎症和气道高反应性是哮喘的三大重要特征。
Accumulation of airway inflammatory cells, chronic airway inflammation and airway hyperresponsiveness are three important features of asthma.
阿司匹林可以影响炎症时涉及的（炎性）复合物和哮喘者存在的气道高反应性。
Aspirin can affect compounds involved in inflammation and the hyper-response of airways that is seen in asthma.
结论：豚鼠cva模型的气道高反应性、病理改变均低于豚鼠哮喘模型，并高于正常对照组。
Conclusion: CVA model of guinea pig airway responsiveness in pathological changes were lower than the level measured in guinea pig asthma model, and higher than the normal control group.
结论：RSV感染导致气道副交感神经自身抑制性M2受体功能障碍，引起气道高反应性发生。
CONCLUSION: These results indicate that RSV infection can cause dysfunction of M2 autoreceptor on the airway preganglionic cholinergic nerves, resulting in airway hyperresponsiveness.
引起气道高反应性的潜在因素包括变应原，锻炼，冷空气，病毒感染，某些药物，或气道的机械刺激。
Potential stimulants of airway hyperreactivity may include allergens, exercise, cold weather, viral infections, medicines, or mechanical airway stimulation.
结论PI 3 K信号通路可能通过促进气道平滑肌细胞增殖参与哮喘气道高反应性及气道重构过程。
Conclusion the PI3K signal pathway may be involved in AHR and the airway remodeling through promoting airway smooth muscle cells proliferation in asthma.
背景与目的哮喘是慢性气道免疫性炎症，以气道淋巴细胞、嗜酸性粒细胞浸润、气道高反应性、粘液产生增加为特征。
Background and Objective Asthma is chronic airway immune inflammation characterized by airway lymphocyte infiltration and eosinophilia, airway hyperresponsiveness (AHR) and mucus hypersecretion.
结论哮喘组大鼠肺组织活性氧产量增加。MT干预可以降低肺组织活性氧产生，降低气道炎症和气道高反应性，这可能是其治疗哮喘的保护机制。
Conclusions MT can decrease airway inflammation and the content of ROS in asthmatic rats, which may be the underlying protective mechanisms of MT against asthma.
结论哮喘组大鼠肺组织活性氧产量增加。MT干预可以降低肺组织活性氧产生，降低气道炎症和气道高反应性，这可能是其治疗哮喘的保护机制。
Conclusions MT can decrease airway inflammation and the content of ROS in asthmatic rats, which may be the underlying protective mechanisms of MT against asthma.
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