左图显示FTO肥胖基因携带者大脑中存在某些缺陷。
The image at left reveals some brain deficits in people with the FTO obesity gene.
环境研究人士将这些化学卡路里称为“肥胖基因”。
Environmental researchers call these chemical calories "obesogens."
他们还发现，有任何肥胖基因的女性也更有可能提前进入青春发育期。
They have also discovered that women with any of the obesity genes are also more likely to enter puberty prematurely.
而32组“肥胖基因”在人体体质指数变化中只占1.45%的比例。
The 32 known "obesity genes" only account for 1.45 percent of the variation in BMI.
瘦素是肥胖基因编码的产物，可调控进食行为和能量消耗。
Leptin, the product of the adipose specific ob gene, regulates food intake and energy expenditure.
贪杯的胖小伙们一定乐意把天生有肥胖基因看成是很好的借口。
You're stuck with the genes you're born with, which could be just the excuse lardy lads who like a drink are looking for.
目前大多数研究的重点都是寻找与食欲或热量消耗有关的肥胖基因。
Much of the recent research focus has been on the intense hunt for obesity genes involved in appetite or calorie burning.
瘦素是肥胖基因的表达产物，通过与其受体结合发挥其生物学作用。
The leptin is a expression product of the obesity gene. Biological effect of leptin-has appeared by combination with its receptor just.
目的探讨胎盘肥胖基因表达与脐血瘦素水平和胎儿宫内生长发育的关系。
Objective To study the role of placental leptin in intrauterine cord leptin production and its relationship with neonatal anthropometry.
这项研究支持了科学家们提出的肥胖基因与结肠癌之间有基因连带关系的最初观点。
The study provides what scientists say is the first evidence of a genetic link between a fat cell gene and colen colon cancer.
瘦素是肥胖基因编码的蛋白相关产物，主要由脂肪组织分泌，调节脂肪代谢和能量平衡。
Leptin is the protein-related product encoded by obesity gene. It is mainly secreted by adipose tissue and can regulate metabolism of adipose and balance of energy.
肥胖基因作用于控制食欲和饱腹感的下丘脑。肥胖基因将增加能量的摄取，特别是脂肪。
The FTO genes ACTS in the hypothalamus, the part of the brain that regulates appetite and satiety, and the risk variant has been connected to an increased energy intake, especially in the form of fat.
瘦素是由肥胖基因编码的一种分泌型的蛋白质，其作用主要在于调节体重、脂肪和能量代谢。
Leptin is a secretory protein encoded by obese gene, which plays role in regulating weight, fat and energy metabolisms.
肥胖基因将增加能量的摄取，特别是脂肪。这可能由于携带者并不会感觉到吃了足够脂肪，因此吃了过多而肥胖。
“It could be that the carriers of the risk gene don’t feel as full from eating fat and therefore consume more and gain weight” says Emily Sonestedt.
报告中说明好几项研究发现，运动消弱了肥胖基因的作用，而且这是第一次在基因的影响和饮食习惯之间做相关研究。
Several studies have found that exercise diminishes the effect of the risk gene but this is the first study where the effect of the gene has been studied in relation to food habits.
出生时带有所谓肥胖基因的孩子并非注定要肥胖，但是科研人员说，这些孩子必须付出额外的努力使体重保持在健康水准。
Children born with the so-called fat genes are not doomed to become obese, but the scientists said those children will have to work extra hard to keep their weight at a healthy level.
王肯恩与他的团队正在调查青春期提前与会引起肥胖的特定基因变异之间的关系。
Ken Ong and his team are investigating the relationship between early puberty and specific genetic variants that can cause obesity.
但是到现在为止，遗传学者仅发现少量的的基因突变导致肥胖的孤立案例。
However, so far, geneticists have found only rare gene mutations that contribute to isolated cases of obesity.
研究员克里斯·丘奇说：“我们第一次提供了令人信服的证据，证明F TO基因导致肥胖。”
Researcher Chris Church said: 'For the first time we have provided convincing proof that the FTO gene causes obesity.
研究人员证实，一种与肥胖有关的流氓基因能通过促进食欲，使我们发胖。
Researchers have shown that a rogue gene linked to obesity makes us fat by boosting appetite.
肥胖是由基因，化学物质，行为和环境共同影响的复杂态，想要解释它需要一个综合的，全面的途径。
Because obesity is a complex condition affected by genes, chemicals, behavior, and the environment, explaining it will likely require a comprehensive approach.
另一组人学习能够导致肥胖的基因和环境因素，例如家族历史和垃圾食品的促销。
Another group learned about the genetic and environmental factors that can contribute to obesity, such as family history and junk food marketing.
肥胖具有基因根源的事实并没有解开更大的谜团。
The fact that obesity has genetic roots does not explain the largermystery.
癌症和心脏病的发生是多个原因的，例如，基因、吸烟、肥胖和饮食，所以不是说简单地服用复合维生素就可以预防。
Both cancer and heart disease are conditions with multiple causes such as genetics, smoking, obesity and diet, so it is unlikely that simply taking a multi-vitamin supplement would prevent them.
相反地，为什么家猫会变得肥胖和懒惰，而和它接近基因的野猫却很健康。
Conversely, why does the house cat grow obese and lethargic, while a bobcat with nearly identical genes stays fit?
肥胖雄鼠的雌性后代身上600个以上的胰岛出现了基因表达的改变。
The daughters of the obese male rats showed an altered expression of more than 600 pancreatic islet genes.
为此，他们研究因为罕见的基因突变而自然肥胖的老鼠。
To find out, they studied mice that were naturally obese because of a rare genetic mutation.
为此，他们研究因为罕见的基因突变而自然肥胖的老鼠。
To find out, they studied mice that were naturally obese because of a rare genetic mutation.
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