一个可能的解释与制造ATP的组织释放出的自由基有关,人们认为它通过破坏细胞来促进老化,并且导致这种像癌症一样的和年龄有关的疾病。
One possibility relates to the ATP-making machinery's emission of free radicals, which are thought to contribute to aging and to such age-related diseases as cancer by damaging cells.
确定的第一个有机自由基是三苯甲基。
The first organic free radical identified was triphenylmethyl radical.
氧自由基学说应该是老年性聋的发病病因。
The oxygen radicals theory should be the etiology of presbyacusis.
研究了异氰酸及异硫氰酸与乙烯自由基之间发生的反应。
Reaction of the isocyanic acid (HNCO) and isothiocyanic acid (HNCS) with ethylene radical is studied.
产生的代谢产物是二甲基砷自由基和二甲基砷过氧自由基。
Resulting metabolites are the dimethylarsenic radical and the dimethylarsenic peroxyl radical.
烷基自由基对羰基进行进攻,生成双环的羰自由基中间体。
This radical attacks the carbonyl group to an intermediate bicyclic ketyl.
蛋白质帮助修复被自由基破坏的受损细胞。
Protein helps repair cells that have suffered free radical damage.
那些指标包括炎症,胰岛素抵抗力和氧自由基。
Those markers include inflammation, insulin resistance and oxygen free radicals.
抗氧化剂与自由基融合,使自由基失去攻击能力。
Antioxidants will merge with these free radicals, turning them harmless.
当人体分解食物时,会产生一种叫做自由基的化合物。
When your body breaks down food it produces molecules called free radicals.
高浓度的自由基会损伤包括晶状体细胞在内的各种细胞。
High levels of free radicals can damage cells, including those in the lens of the eye.
氧自由基吸收价值是衡量产品对抗自由基损害细胞的能力。
The ORAC value is a measure of the capacity of the product to subdue free radicals that damage cells.
如前所述,退化性疾病由于自由基持续对我们细胞的损害。
As stated earlier, degenerative illness is a function of free radical damage to our cells over time.
自由基被认为是心脏病,癌症和老化过程中重要的第一步。
Free radicals are believed to be a first important step in heart disease, cancer and the ageing process.
随着年龄的增长,浮游在脑血管中的自由基会摧毁脑细胞。
As people get older, substances called free radicals, which are floating through the bloodstream, will break down the brain cells.
他们也知道抗氧化剂能中和自由基,理论上能使它们不那么危险。
They also know that antioxidants can stabilize free radicals, theoretically making them much less dangerous.
这些抗氧化剂还能够对抗大脑内及其它身体各部位的自由基损伤。
These antioxidants also fight against free radical damage that can occur within the brain as well as the body.
科威特大学的科学家们,致力于寻找大蒜是否能减少自由基受损。
Scientists at the University of Kuwait were interested in finding out whether garlic could reduce free radical damage.
科学家们检查(DADS)能够在产生自由基的过程中杀灭细胞。
Scientists there examined DADS ability to kill cells in a process involving free radical production.
自由基是一种能破坏身体细胞的分子物质,其最主要的来源就是糖。
Free radicals are molecules that destroy body cells. A major source is thought to be sugar.
吸烟的过程会产生一种叫做自由基的化学物质,从而损伤眼内的晶体。
Smoking may damage the lens of the eye by leading to the formation of chemicals called free radicals.
碱类物质也可以承载抗氧化剂物质,协助抵抗可能会导致疾病的自由基。
And phenols are also loaded with antioxidants which help to battle free radicals that can cause disease.
证据显示,太快地吸入过多的氧气可能夺走单电子的分子,形成自由基。
Evidence suggests that pumping in too much oxygen too quickly can strip the molecule of a single electron, creating a free radical.
鱼油会很快分解,分解过程中释放出自由基,而自由基与细胞损害有关。
Fish oils decompose quickly and in the process unleash free radicals, which are linked to cell damage.
而且,该研究只表明自由基可以延长寿命,并没有说明对生活质量的影响。
Also, the study only shows the effects of free radicals on longevity, and can say nothing about quality of life.
交通污染物质中往往含有更多的金属物质,可能在肺部产生有害的自由基。
Traffic pollution tends to contain more metals, which react to produce damaging free radicals in the lungs.
基因变异的虫子向我们演示,自由基可以帮助触发身体的保护和修复机制。
The genetically modified worms demonstrated that the production of free radicals can help to trigger the body’s general protective and repair mechanisms.
增加精索可以抑制各种与衰老有关的过程,还能减少自由基并延长生命跨度。
Adding spermidine suppressed various processes associated with ageing, as well as reducing free radicals and increasing lifespan.
但是如果我们通过控制自由基的信息和释放来干预基因的表达,结果会怎样?
But what if we intervene on behalf of gene expression by controlling the formation and liberation of free radicals in the body?
如果有人携带关节炎的基因,那么基因编码可能将自由基储存在关节组织里。
If someone carries the gene for arthritis, for example, one would expect genetic coding to foster storage of free radicals in their joint tissues.
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