它被称为髓磷脂。
测试的每种老鼠，不论缺少是哪种菌株，他们发现在出生后六周后出现了髓磷脂损害的现象。
In every mouse they tested, regardless of strain, they found early evidence of myelin damage just six weeks after birth.
髓磷脂是很难看到的。它就象模模糊糊的、起伏波动的饰缘镶在神经元的边缘上。
The myelin was harder to see, a faintly undulating fringe on the edge of the neurons.
得不到充足的这种原料的婴儿，会在制造帮助神经元传导信号的含脂肪的髓磷脂绝缘层时遇到问题。
Babies who don't get enough of the stuff have trouble creating the fatty myelin insulation that helps neurons transmit signals.
正象巴兹·克斯所说的那样：“髓磷脂是我们的阿基里斯的力量，它是我们的脚跟。”
As Bartzokis had said: "Myelin is our Achilles strength, and it's our Achilles' heel."
髓磷脂的损失也是多发性硬化的原因，并在诸如糖尿病与高血压等其它疾病也进展到一定的程度。
Myelin loss is also the reason for multiple sclerosis, and occurs to some extent in other diseases like diabetes and high blood pressure.
丰富的髓磷脂在她们大脑的右侧，对于右侧的肌肉群来说髓磷脂可以起到优化她们神经电路的作用。
They've got more in the right parts of the brain and for the right muscle groups, and that's what allows them to optimize their circuitry.
拿小孩子来说，髓磷脂是以一系列波的形式到达，其中一部分有生物密码决定，另一部分则依靠练习活动。
In children, myelin arrives in a series of waves, some of them determined by biological code, some of them dependent on activity.
因为老鼠在出生时没有髓磷脂损害，我们假设这种蛋白是为了维持髓磷脂的质量，并且在整个生命周期内会逐渐减小。
"Because there is no myelin damage at birth, we assumed prions are needed to maintain the quality of the myelin sheath, which diminishes throughout life," says Aguzzi.
髓磷脂不足或工作状态差可导致一系列疾病，包括影响几千个孩子的一种罕见与致命的遗传病：小儿脑白质营养不良。
Insufficient or poorly working myelin causes a number of diseases, including a rare and fatal congenital disorder called pediatric leukodystrophy that affects thousands of children.
目的探讨髓磷脂碱性蛋白mBP刺激的PBMC培养上清作用的人脐静脉内皮细胞HUVEC对T细胞增殖的影响。
Aim To explore the effect of human umbilical vein endothelial cell (HUVEC) co incubated with the culture supernatant of MBP stimulated PBMC on the t cell proliferation.
为了了解更多，这个团队又观察了一岁大的老鼠们，他们发现老鼠的一根从背部走向腿部的大神经，也就是坐骨神经里的髓磷脂渐渐丢失。
To learn more, the group looked at 1-year-old mice and found that their sciatic nerve, the large nerve in the back that runs into the legs, had lost myelin.
Fingolimod通过阻止免疫细胞攻击髓磷脂而起作用。髓磷脂是一种脂肪能够隔离神经纤维从而帮助他们在大脑和躯体之间传导信号。
Fingolimod works by preventing immune cells from attacking myelin, the fatty substance that insulates nerves and helps them transmit impulses between the brain and the rest of the body.
脊髓横断组髓磷脂碱性蛋白（MBP）阳性的少突胶质细胞数目的时间及空间分布与正常对照组相比无统计学差异（P>0.05）。
The number of myelin basic protein (MBP) immunopositive oligodendrocytes in SCI groups was not obviously different from that in the control group (P>0.05).
目前，一组科学家说，PrP在供养髓磷脂中发挥重要作用，髓磷脂是多脂肪的物质形成的鞘，这个鞘存在于神经周围，用以传递神经发出的信号。
A team of scientists now says that PrP plays a key role in maintaining myelin, a fatty substance that forms a sheath around nerves and helps transmit nerve signals.
目前，一组科学家说，PrP在供养髓磷脂中发挥重要作用，髓磷脂是多脂肪的物质形成的鞘，这个鞘存在于神经周围，用以传递神经发出的信号。
A team of scientists now says that PrP plays a key role in maintaining myelin, a fatty substance that forms a sheath around nerves and helps transmit nerve signals.
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