高铁血红蛋白还原酶途径利用了NADH的还原电位维持血红蛋白中铁元素的还原态(Fe2 +)。

The reducing potential of the NADH is utilized via the methemoglobin reductase pathway to maintain the iron in Hgb in its reduced form (Fe2 +).

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过大的氧化压力可能使磷酸己糖支路或高铁血红蛋白还原酶途径不堪重负，分别造成海因茨小体性溶血或形成高铁血红蛋白。

Excessive oxidant stress may overload the protective HMP shunt or methemoglobin reductase pathways, causing Heinz body hemolysis or methemoglobin formation, respectively.

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