脑缺血再灌注损伤（Brain ischemia and reperfusion injury，BIRI）是由多种机制参与的由于脑缺血及再灌注导致的病理性过程，其中与活性氧自由基（reactive oxyg...
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Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Objective:To investigate whether brain surface cooling(BSC) can decrease cerebral content of water and Evans Blue(EB) after ischemia-reperfusion injury in experimental rabbits.
目的：探讨脑表面降温对缺血再灌注损伤后的脑组织含水量和伊纹氏蓝含量的影响。
Conclusion DG can markedly inhibit nerve cell apoptosis induced by focal brain ischemia reperfusion injury and protect the nerve cell to a certain extent.
结论DG可明显抑制局灶性脑缺血-再灌注所诱导的脑神经细胞凋亡，起到一定的神经保护作用。
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