Serum concentrations of glucose, glycosylated serum protein, and malondialdehyde, derived from lipid peroxidation were also examined to estimate metabolic control.

并检测血糖、糖基化血清蛋白和血清脂过氧化产物丙二醛浓度以反映代谢控制。

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Inhibiting lipid accumulation in macrophage-derived foam cells and reduce inflammatory responses may be of therapeutic value in preventing coronary artery disease.

抑制巨噬细胞源泡沫细胞中脂质聚积、减轻炎症反应可能有动脉粥样硬化预防、治疗价值。

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Conclusion: the protective mechanisms of YQTLP were related to inhibit lipid peroxidation, protect endothelium-derived relaxing factor and adjust the fibrinolytic activities.

结论：益气通络丹的疗效机制与抑制脂质过氧化、保护内皮依赖性松弛因子和调节纤溶活性有关。

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