目的探讨角膜基质细胞组织因子途径抑制物2 (TFPI - 2)的表达情况，为角膜新生血管的基因治疗提供方法。
Objective to observe the expression of TFPI-2 in corneal keratocytes in vitro and investigate the experimental basis for curing corneal neovascularization with gene-therapy.
如图所示为严重化学伤后早期发生的角膜新生血管形成。
The image shown demonstrates the effects of a severe chemical injury with early corneal neovascularization.
以SD角膜新生血管化大鼠为受体，建立大鼠高危穿透性角膜移植动物模型。
SD rats with corneal vascularization were taken as recipients to establish animal model of high-risk penetrative corneal transplantation.
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