目的探讨一氧化氮（NO）在缺血预处理（IP）保护大鼠移植肝脏缺血再灌注损伤中的作用。
Objective To investigate the protective effect of nitric oxide (NO) induced by ischemic preconditioning (IP) on ischemic reperfusion injury of rat liver graft.
在缺血-再灌注损伤前24小时进行缺血的预处理可增加VEGF - A水平并大量减少凋亡的视网膜细胞数量。
Ischemic preconditioning 24 hours before ischemia-reperfusion injury increased VEGF-A levels and substantially decreased the number of apoptotic retinal cells.
目的：研究内源性一氧化碳（CO）在大鼠肝脏缺血预处理中的保护作用机制。
Objective:To ascertain whether CO/cGMP pathway is invovled in the protective mechanism of Endogenous carbon monoxide in the rat liver undergone ischemic preconditioning.
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