胰岛素信号转导途径目前已知至少有两条：一是通过胰岛素受体底物蛋白（insulin receptor substrate，IRS）激活磷脂酰肌醇3激酶（phosphatidylinositide-3kinase，PI-3K）途径；...
基于4个网页-相关网页
对丁酸钠转化前后的人红白血病细胞林k - 562的表面胰岛素受体数量、胰岛素受体酪氨酸蛋白激酶活性及细胞内源性底物进行了研究。
In this paper, the quantity of insulin receptors and the receptor tyrosine kinase activity and its endogenous substrates were comparatively studied in K-562 cells before and after transformation.
本文对丁酸钠诱导分化的人白血病细胞株k - 562细胞的胰岛素受体酪氨酸蛋白激酶性及细胞内源性底物进行了研究。
In this paper, the tyrosine protein kinase activity of insulin receptor and its endogenous substrates were investigated on the human leukemia cell line K-562 differentiated by sodium butyrate.
通过减少胰岛素受体底物（IRS）蛋白的水平能够增加胰岛素受体活性时间、炎症时间和胰岛素水平，从而有助于降低胰岛素抵抗程度。
Prolonged activation of the insulin receptor, inflammation, and excessive insulin levels can induce insulin resistance by decreasing levels of insulin receptor substrate (IRS) proteins.
应用推荐