雌激素受体基因是一种对骨密度有调控作用的基因。
Er gene has been suggested as a gene for regulation of bone mineral density (BMD).
结论：染料木黄酮不是通过促进雌激素受体基因和蛋白的表达来促进成骨细胞增殖。
CONCLUTION Genistein promotes the proliferation of osteoblast not through promoting the expression of estrogen receptor gene and protein.
分析雌激素受体基因多态性与骨密度的关系及各基因型在骨质疏松组与对照组的分布。
Pvu II and Xba I restriction fragment length polymorphisms (RFLP) of ER were analysed by polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP) .
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