To investigate BCL11A's therapeutic value, Orkin's team turned to a mouse model for sickle cell disease.
为了进一步研究BCL11A的治疗价值,奥尔金的研究小组转向镰状贫血症的白鼠模型。
The researchers genetically blocked ERBB3 in a mouse model of colon cancer and in human colon cancer cell lines.
研究者们从遗传上敲除了结肠癌鼠模型和人类结肠癌细胞系的ERBB3基因。
When they tested drugs that block two of these proteins in a mouse model of multiple sclerosis, the mice improved dramatically.
在多发性硬化小鼠模型的身上,他们用药物阻断这些蛋白质中的两种蛋白,病变小鼠得到显著改善。
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