The researchers tested different bitter compounds on human and mouse airways, individual airway smooth muscle cells, and on mice with asthma.

研究人员将苦味化合物放在不同的试验组进行测试：人类及老鼠的气管、个人气管平滑肌细胞、哮喘的小老鼠。

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CONCLUSIONS: HCY induced proliferation of airway smooth muscle cells and fibroblasts, HCY also induced collagen production of airway fibroblasts.

结论：HCY刺激气道平滑肌细胞及成纤维细胞增殖，并促进成纤维细胞的胶原合成和分泌。

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Conclusion the PI3K signal pathway may be involved in AHR and the airway remodeling through promoting airway smooth muscle cells proliferation in asthma.

结论PI 3 K信号通路可能通过促进气道平滑肌细胞增殖参与哮喘气道高反应性及气道重构过程。

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