AD的三大病理特征是患者脑内出现以13．淀粉样蛋白（amyloid beta peptide，AD）为主要成分的细胞外老年斑（senileplaque，SP）、细胞内神经原纤 维缠结（neurofib...
基于1个网页-相关网页
Beta-Amyloid Peptide 毒性物质
Researchers have confirmed that incidence of AD was due to the aggregation of amyloid beta peptide (Aβ). Being the hydrophobic core of Aβ, Aβ16-22 possesses important research value.
由于AD的发病是淀粉质多肽(Aβ)聚集的后果,而Aβ16-22又是Aβ聚集的重要疏水性核心,因此具有重要研究价值。
参考来源 - 海藻糖抑制淀粉质β多肽聚集的实验研究·2,447,543篇论文数据，部分数据来源于NoteExpress
One of the products, with the long-winded name amyloid beta-peptide 42, has the unfortunate property that its molecules like to stick to each other.
所形成的产物之一有一个冗长的名字——淀粉样贝塔-肽42，它具有一种不祥的特性，即它们分子彼此之间容易纠缠在一起。
For the past decade there has been a lot of work on drugs called secretase inhibitors, which alter the activity of the enzymes that create amyloid beta-peptide 42.
在过去的10年里人们对被称为分泌酶抑制剂的药物进行了大量研究，这种药物可以改变酶类产生淀粉样贝塔-肽42的活动。
The NYU researchers stitched together the string of amino acids on amyloid beta to create their synthetic peptide "mimic."
NYU的研究员将β淀粉状蛋白的氨基酸链整合以创造出模仿的合成肽。
应用推荐