People with the disease get plaques in their brains made up of a small protein called amyloid-beta, which clumps together and disrupts brain signals.

得了这种病的人大脑里面的血小板形成一种叫做淀粉样蛋白的蛋白质，这种蛋白质聚集在一起打乱了大脑发出的信号。

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He has tested it so far in mice, and found the animals have better memory and other cognitive skills and fewer clumps of beta-amyloid and tau protein.

目前的实验对象为老鼠。他还发现动物具有更强的记忆力和其他认知能力，更少的贝塔淀粉样和tau蛋白质凝结。

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Amyloid plaques form when a molecule called amyloid precursor protein (APP) is chopped up by two enzymes known as beta-secretase and gamma-secretase.

当一种叫淀粉样前体蛋白（APP）的分子被两种酶——分别叫作贝塔-分泌酶和伽玛-分泌酶——切断时就形成了淀粉样斑块。

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To be specific, Merck (MRK) will test a beta-amyloid precursor protein site-cleaving enzyme, or BACE, inhibitor, which the drugmaker notes is the first drug with this type of mechanism to advance to this stage of clinical research.

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Most researchers are focusing on blocking the formation of amyloid-beta protein or removing it from the brain, either before or after deposits are formed.

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Harvard University researchers conducted experiments on mice using oligomers, a soluble form of amyloid-beta protein, which is the key compound in brain plaque and a hallmark of Alzheimer's.

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