In experiments on mice, researchers tracked brain levels of cell-signaling protein molecules called cytokines, which are boosted by SSRIs, and found they were counteracted by anti-inflammatory drugs.
研究人员在白鼠上实验，追踪叫做细胞活素类的细胞信号传导蛋白的脑部水平(它受ssris的刺激)，发现消炎药削弱了这些蛋白质的活动。
The expressions of many genes encoding transcription factor, cytokines, cell signaling modulators and apoptosis associated proteins were found to have changed.
许多转录因子、细胞内信号转导调节蛋白、炎症细胞因子和细胞凋亡相关基因的表达均发生了明显的调节变化。
Without miR-155, they were unable to generate important cytokines, the cell-to-cell signaling proteins that coordinate the various components of the immune system.
没有miR - 155，它们无法产生重要的细胞因子，细胞因子是细胞和细胞间的信号蛋白，可以协调免疫系统各个部分。
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