Using the techniques they developed, researchers discovered that TLR4 proteins cluster in the membrane when confronted with LPS derived from e. coli, which increases cell signalling and response.
通过使用这种成像技术，研究人员发现TLR4蛋白质遇到来自大肠杆菌的脂多糖时会聚集在细胞膜内，提高细胞发出信号和反应的能力。
Rats were challenged by E. coli LPS to set up the model of endotoxic shock, AG group were treated by aminoguanidine.
用大肠杆菌内毒素（LPS）复制大鼠内毒素性休克模型，氨基胍治疗组采用氨基胍治疗。
The effect of E. coli endotoxin (LPS) on early and advanced experimental cirrhosis have been studied.
本文对大肠杆菌内毒素(LPS)在实验性早期和晚期肝硬化中的致病作用进行了研究。
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