The molecule blocked a gene that codes for a receptor for GABA, a brain signaling molecule.

该分子抑制了产生氨基丁酸受体这种大脑内信号分子的基因片段。

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This finding suggests ways to improve anxiolytics and to develop drugs for other neurological disorders based on their specificity for GABA(A) receptor subtypes in distinct neuronal circuits.

这个发现指明了改善舒缓焦虑的方法，并得以为在不同的神经元回路中，特定之GABA（A）受体子类的其他神经方面失调，开发新药。

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These results indicated that GABA receptor expressed in Xenopus oocyte membrane is not type B but type A.

基于上述结果，本实验根据神经系GABA受体亚基多肽链第二跨膜区的。

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But all those drugs work by variations on the same brain mechanism, hitting the same GABA-A receptor that makes valium sedating.

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