hypoxia-ischemia cerebral damage 缺血缺氧脑损伤
Conclusion Propofol may inhibit hypoxia in the brain and the apoptosis of nerve cells in result of protecting the cerebral ischemia and reperfusion damage in rats.
结论异丙酚可能通过抑制大脑缺氧，抑制神经元的凋亡，从而对大鼠局灶性脑缺血-再灌注损伤发挥保护作用。
This article introduces the effect of high central nervous system, changes of cerebral blood flow after hypoxia, damage of hypoxia to neurons in the respiratory center, neurotransmitters a…
本文主要介绍高级中枢、缺氧后脑血流量变化、缺氧对呼吸中枢神经元的实质性损害、 神经递质和神经调质在缺氧性呼吸抑制过程中的作用。
Apoptosis was detected with flow cytometry after hypoxia-reperfusion oxygen damage of the cerebral microvascular endothelial cells.
脑微血管内皮细胞缺氧复给氧损伤后，采用流式细胞仪检测细胞凋亡的程度。
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