In the study group, pulmonary architecture was preserved and infiltration of inflammatory cells and edema decreased.
与对照组相比较,各时间点的实验组大鼠肺组织炎症细胞渗出减少,间质水肿减轻。
The pathological changes of the esophagus and fore stomach consisted of exfoliation of the squamous epithelium and infiltration of inflammatory cells.
食管及前胃的组织病理学改变以鳞状上皮脱落及炎症细胞浸润为主。
Conclusions Long-term smoking may result in the infiltration of inflammatory cells in the bronchiolar walls and the morphology changes of lung tissues, leading to COPD.
结论长期吸烟可引起气道壁炎细胞浸润及肺组织形态学变化,导致COPD发生。
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