human lung adenocarcinoma cells A549 人肺腺癌细胞株A
Objective To investigate the roles of PI3K pathway in multicellular resistance of lung adenocarcinoma A549 cells.
目的探讨PI3K信号通路在肿瘤群集耐药中的作用。
Conclusion The overexpression of CC10 might play an important role in the matrine-induced apoptosis of lung adenocarcinoma A549 cells.
结论CC10基因表达增高可能在苦参碱诱导肺腺癌A549细胞凋亡中起重要作用。
AIM: to study the apoptosis of human lung adenocarcinoma A549 cells induced by CN-2, a derivative of podophyllotoxin, and to obtain insights into its molecular mechanism of action.
目的研究鬼臼素衍生物cn - 2对人肺腺癌细胞A549的凋亡诱导作用，并对其分子机制进行初步探讨。
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