Conclusion CACNA1H gene G773D mutation is able to increase calcium current and neuronal excitability.

结论CACNA1H基因G 773d突变可使其编码通道电流增加，并可能引起神经元兴奋性增加。

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Previous electrophysiological experiments have revealed that melatonin modulates hippocampal neuronal excitability and plasticity.

已有的电生理学实验已经表明，褪黑素可以调节海马神经元的兴奋性和突触可塑性。

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Conclusions: These results indicate that NOS is extensively distributed in hippocampal cells and its activation leads to an increase of the neuronal excitability.

结论：以上结果显示NOS广泛分布在海马神经细胞中，当其激活时对海马神经元有兴奋作用。

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