The production of nitric oxide(NO) was determined by nitrate reductase method.
用硝酸还原酶法测一氧化氮（NO）的浓度。
Adenosine stimulated a significant arteriolar nitric oxide (NO) production and dilation, which were sensitive to NO synthase inhibitor L-NMMA.
腺苷能够显著激发动脉一氧化氮（NO）的产生而使血管舒张，且对一氧化氮合酶抑制剂L - NMMA 十分敏感。
The results demonstrated that nifedipine can inhibit the production of nitric oxide in T629 cells, CPAE cells and SM3 cells, and also inhibit the amount of NO stimulated by LPS in macrophages.
结果表明硝苯地平能够有效抑制上述细胞一氧化氮的释放，且抑制效应呈剂量依赖关系。
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