【目的】探讨压力负荷型心肌肥厚(pressure-overload myocardial hypertrophy，POH)、肥厚-逆转(POH-Reverse，POH-Re)、慢性心力衰竭(chronic congesti...
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Conclusion Under chronic pressure overload, the activated ASK1 with the increasing ROS may play an important role in signal transduction pathway of myocardial hypertrophy.
结论压力超负荷下随着ROS的升高，激活的ASK1可能在心脏肥大信号传导途径中发挥重要作用。
Objective: To study the role of nitric oxide in myocardial hypertrophy caused by pressure overload in rats.
目的：探讨一气化氮在压力超负荷心肌肥大反应过程中的作用。
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