We hypothesized that alterations in cellular protein S-nitrosylation may contribute to hyperglycemia-induced endothelial dysfunction.
我们假设细胞蛋白s -亚硝基化的变化会引起高血糖介导的内皮功能障碍。
One area is S-nitrosylation, where we've been doing some work with a group at Johns Hopkins, the protein, transglutaminase2, seems to be effected by this process S-nitrosylation.
其中之一就是S-亚硝基化，我们已经和霍普金斯的一个研究组合作了一些研究，结果一种蛋白，谷氨酰胺转移酶2，似乎能被这一S-亚硝基化过程所影响。
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